ER Stress is Linked to the Regulation of Hepatic Gluconeogenesis and Phosphatidylinositol 3-kinase/Akt Signaling Post Burn Injury

Burn injury is a severe form of trauma that causes changes in glucose metabolism. Glucose metabolism is controlled during early burn injury but later results in stress induced diabetes. We have shown that this is due to differential regulation of the insulin signaling pathway in the liver.

Name thesis:degree_name

Biochemistry and Molecular Biology (Doctoral)

Level thesis:degree_level

Doctoral

Discipline thesis:degree_discipline

Molecular Biology

Grantor

The University of Texas Medical Branch at Galveston

Author dc:creator

• Brooks, Natasha 1983-

Advisor dc:contributor.advisor

• Perez-Polo, J. Regino

Committee members dc:contributor.committeemember

• Jeschke, Marc G
• Boehning, Darren
• Tilton, Ronald
• Aubert, Isabelle

• Burn injury
• ER Stress
• glucose metabolism

Handle dc:identifier.uri

http://hdl.handle.net/2152.3/841

OAI identifier oai:identifier

oai:utmb-ir.tdl.org:2152.3/841