{"id":{"repo_id":"uiuc","oai_identifier":"oai:www.ideals.illinois.edu:2142/85054"},"canonical_url":"https://search.dev.ndltd.org/etd/uiuc/oai:www.ideals.illinois.edu:2142/85054","repository":{"repo_id":"uiuc","name":"University of Illinois - Urbana-Champaign","base_url":"https://www.ideals.illinois.edu/oai-pmh"},"display":{"title":"Inheritance of Aspergillus Ear Rot and Aflatoxin Resistance From the Tex6 Corn Line","abstract":"F$\\sb1$ means deviated from the midparent value toward resistance for aflatoxin production and toward susceptibility for ear rot in both crosses. In the B73 $\\times$ Tex6 cross, generation mean analysis indicated additive gene action was the most important type of gene action controlling resistance to ear rot and to aflatoxin production. Mo17 was less susceptible to both traits, so the resistance from Tex6 in crosses with Mo17 was not well defined. Broad-sense heritabilities were 58% for ear rot and 63% for aflatoxin production in the Mo17 $\\times$ Tex6 cross, and 66% for ear rot and 65% for aflatoxin production in the B73 $\\times$ Tex6 cross. Narrow-sense heritabilities were 39% for ear rot and 45% for aflatoxin production in the B73 $\\times$ Tex6 cross. Expected gain from F$\\sb3$ family selection was 11.1% for ear rot and 1.5% for aflatoxin production. Selection will be more effective within F$\\sb3$ families of the B73 $\\times$ Tex6 cross than F$\\sb3$ families of the Mo17 $\\times$ Tex6 cross, particularly in environments favorable for disease development.","abstract_html":"F$\\sb1$ means deviated from the midparent value toward resistance for aflatoxin production and toward susceptibility for ear rot in both crosses. In the B73 $\\times$ Tex6 cross, generation mean analysis indicated additive gene action was the most important type of gene action controlling resistance to ear rot and to aflatoxin production. Mo17 was less susceptible to both traits, so the resistance from Tex6 in crosses with Mo17 was not well defined. Broad-sense heritabilities were 58% for ear rot and 63% for aflatoxin production in the Mo17 $\\times$ Tex6 cross, and 66% for ear rot and 65% for aflatoxin production in the B73 $\\times$ Tex6 cross. Narrow-sense heritabilities were 39% for ear rot and 45% for aflatoxin production in the B73 $\\times$ Tex6 cross. Expected gain from F$\\sb3$ family selection was 11.1% for ear rot and 1.5% for aflatoxin production. Selection will be more effective within F$\\sb3$ families of the B73 $\\times$ Tex6 cross than F$\\sb3$ families of the Mo17 $\\times$ Tex6 cross, particularly in environments favorable for disease development.","abstract_has_math":true,"creators":["Hamblin, Andrew Maurice"],"institution":"University of Illinois at Urbana-Champaign","degree_name":"Ph.D.","degree_level":"Dissertation","degree_discipline":"Crop Sciences","degree_department":null,"school":null,"contributors":["White, Donald G."],"advisors":[],"committee_chairs":[],"committee_members":[],"year":2015,"date_issued":"2015-09-25T22:33:38Z","date_published":"2015-09-25T22:33:38Z","updated_at":"2026-07-22T22:26:24Z","subjects":["Agriculture, Agronomy"],"languages":["eng"],"rights":[],"rights_urls":[],"identifier_entries":[{"key":"dc:identifier","label":"Identifier","values":["(MiAaPQ)AAI9904475"],"render_values":[{"text":"(MiAaPQ)AAI9904475","href":null,"code":true}]}]},"links":{"outbound_url":"http://hdl.handle.net/2142/85054","outbound_label":"Handle","outbound_source":"dc:identifier"},"metadata_groups":[{"id":"people","label":"People","entries":[{"key":"dc:contributor","label":"Contributor","values":["White, Donald G."]},{"key":"dc:creator","label":"Author","values":["Hamblin, Andrew Maurice"]}]},{"id":"academic_context","label":"Academic Context","entries":[{"key":"dc:date","label":"Dc Date","values":["2015-09-25T22:33:38Z","10000-01-01","1998"]},{"key":"dc:type","label":"Dc Type","values":["text"]},{"key":"thesis:degree_discipline","label":"Discipline","values":["Crop Sciences"]},{"key":"thesis:degree_level","label":"Degree Level","values":["Dissertation"]},{"key":"thesis:degree_name","label":"Degree Name","values":["Ph.D."]},{"key":"thesis:institution_name","label":"Thesis Institution Name","values":["University of Illinois at Urbana-Champaign"]}]},{"id":"subjects_keywords","label":"Subjects and Keywords","entries":[{"key":"dc:subject","label":"Dc Subject","values":["Agriculture, Agronomy"]}]},{"id":"language_rights","label":"Language and Rights","entries":[{"key":"dc:language","label":"Dc Language","values":["eng"]}]},{"id":"identifiers","label":"Identifiers","entries":[{"key":"dc:identifier","label":"Identifier","values":["http://hdl.handle.net/2142/85054","(MiAaPQ)AAI9904475"]}]},{"id":"additional","label":"Additional Metadata","entries":[{"key":"dc:description","label":"Description","values":["F$\\sb1$ means deviated from the midparent value toward resistance for aflatoxin production and toward susceptibility for ear rot in both crosses. In the B73 $\\times$ Tex6 cross, generation mean analysis indicated additive gene action was the most important type of gene action controlling resistance to ear rot and to aflatoxin production. Mo17 was less susceptible to both traits, so the resistance from Tex6 in crosses with Mo17 was not well defined. Broad-sense heritabilities were 58% for ear rot and 63% for aflatoxin production in the Mo17 $\\times$ Tex6 cross, and 66% for ear rot and 65% for aflatoxin production in the B73 $\\times$ Tex6 cross. Narrow-sense heritabilities were 39% for ear rot and 45% for aflatoxin production in the B73 $\\times$ Tex6 cross. Expected gain from F$\\sb3$ family selection was 11.1% for ear rot and 1.5% for aflatoxin production. Selection will be more effective within F$\\sb3$ families of the B73 $\\times$ Tex6 cross than F$\\sb3$ families of the Mo17 $\\times$ Tex6 cross, particularly in environments favorable for disease development.","Made available in DSpace on 2015-09-25T22:33:38Z (GMT). No. of bitstreams: 2 license.txt: 4848 bytes, checksum: 96035ab3f5e1c23cc7138a224ce498bd (MD5) 9904475.pdf: 2270797 bytes, checksum: e63162fdf4264ed04730feb88aae2da2 (MD5) Previous issue date: 1998","Embargo set by: Seth Robbins for item 86335 Lift date: Forever Reason: Restricted to the U of I community idenfinitely during batch ingest of legacy ETDs","Restricted to the U of I community idenfinitely during batch ingest of legacy ETDs","U of I Only","56 p.","Thesis (Ph.D.)--University of Illinois at Urbana-Champaign, 1998."]},{"key":"dc:title","label":"Title","values":["Inheritance of Aspergillus Ear Rot and Aflatoxin Resistance From the Tex6 Corn Line"]}]}],"canonical_facts":{"dc:contributor":["White, Donald G."],"dc:creator":["Hamblin, Andrew Maurice"],"dc:date":["2015-09-25T22:33:38Z","10000-01-01","1998"],"dc:description":["F$\\sb1$ means deviated from the midparent value toward resistance for aflatoxin production and toward susceptibility for ear rot in both crosses. In the B73 $\\times$ Tex6 cross, generation mean analysis indicated additive gene action was the most important type of gene action controlling resistance to ear rot and to aflatoxin production. Mo17 was less susceptible to both traits, so the resistance from Tex6 in crosses with Mo17 was not well defined. Broad-sense heritabilities were 58% for ear rot and 63% for aflatoxin production in the Mo17 $\\times$ Tex6 cross, and 66% for ear rot and 65% for aflatoxin production in the B73 $\\times$ Tex6 cross. Narrow-sense heritabilities were 39% for ear rot and 45% for aflatoxin production in the B73 $\\times$ Tex6 cross. Expected gain from F$\\sb3$ family selection was 11.1% for ear rot and 1.5% for aflatoxin production. Selection will be more effective within F$\\sb3$ families of the B73 $\\times$ Tex6 cross than F$\\sb3$ families of the Mo17 $\\times$ Tex6 cross, particularly in environments favorable for disease development.","Made available in DSpace on 2015-09-25T22:33:38Z (GMT). No. of bitstreams: 2 license.txt: 4848 bytes, checksum: 96035ab3f5e1c23cc7138a224ce498bd (MD5) 9904475.pdf: 2270797 bytes, checksum: e63162fdf4264ed04730feb88aae2da2 (MD5) Previous issue date: 1998","Embargo set by: Seth Robbins for item 86335 Lift date: Forever Reason: Restricted to the U of I community idenfinitely during batch ingest of legacy ETDs","Restricted to the U of I community idenfinitely during batch ingest of legacy ETDs","U of I Only","56 p.","Thesis (Ph.D.)--University of Illinois at Urbana-Champaign, 1998."],"dc:identifier":["http://hdl.handle.net/2142/85054","(MiAaPQ)AAI9904475"],"dc:language":["eng"],"dc:subject":["Agriculture, Agronomy"],"dc:title":["Inheritance of Aspergillus Ear Rot and Aflatoxin Resistance From the Tex6 Corn Line"],"dc:type":["text"],"thesis:degree_discipline":["Crop Sciences"],"thesis:degree_level":["Dissertation"],"thesis:degree_name":["Ph.D."],"thesis:institution_name":["University of Illinois at Urbana-Champaign"]},"updated_at":"2026-07-22T22:26:24Z"}