{"id":{"repo_id":"greece","oai_identifier":"oai:10442/1100"},"canonical_url":"https://search.dev.ndltd.org/etd/greece/oai:10442/1100","repository":{"repo_id":"greece","name":"Greek National Archive of PhD Theses","base_url":"https://phdtheses.ekt.gr/eadd_oai/request"},"display":{"title":"ΝΕΥΡΟΦΥΣΙΟΛΟΓΙΚΗ ΜΕΛΕΤΗ ΤΟΥ ΠΕΡΙΦΕΡΙΚΟΥ ΝΕΥΡΙΚΟΥ ΚΑΙ ΜΥΙΚΟΥ ΣΥΣΤΗΜΑΤΟΣ ΣΕ ΑΣΘΕΝΕΙΣ ΜΕ ΟΜΟΖΥΓΟ Β-ΘΑΛΑΣΣΑΙΜΙΑ","abstract":"WE PERFORMED EMG STUDIES IN 53 PATIENTS, TREATED DURING THE PAST FIVE YEARS IN A UNIVERSITY THALASSEMIA UNIT AND IN 29 CONTROLS OF THE SAME AGE RANGE (5 TO 35YEARS). WE STUDIED SEPARATELY SEVEN THALASEMIC PATIENTS WITH THE COMPLICATION OF DIABETES MELLITUS. FROM THE DETAIL CLINICAL EXAMINATION (MEDICAL HISTORY ANDPHYSICAL EXAMINATION) IT WAS FOUND THAT 23% OF THE PATIENTS PRESENTED WITH SENSORY SYMPTOMS (NUMBNESS AND TINGLINGS) IN THE LOWER LIMBS, 17% WITH CRAMPS AND 15% WITH MUSCULAR PAINS. FURTHERMORE 13% OF THE PATIENTS HAD DIMINISHED KNEE JERK AND 19% THE ACHILLES TENDON REFLEX. IN 4% OF PATIENTS THE ACHILLES REFLEX WAS ABSENT. MOTOR CONDUCTION VELOCITIES (MCV) FOR THE DEEP PERONEAL AND THE TIBIAL NERVE WERE REDUCED IN THE PATIENT GROUP COMPARED WITH THE CONTROL GROUP (49.13.37 M/S, SE-0.46 AND 52.2 2.79 M/S, SE=0.51 T-TEST 4.49 FOR THE PERONEAL NERVE AND 48.7 4. 41 M/S, SE=0.56 AND 51.65 3.23 M/S, SE= 0.60 T-TEST 3.58 FOR THE TIBIAL NERVE, RESPECTIVELY). THE GROUP OF THE PATIENTS WITH THE LOWER VALUES OFMCV, WAS THE ONE WHICH SHOWED THE CLINICAL SYMTOMS AND SIGNS, OF PERIPHERAL NEUROPATHY. IN THE GROUP WITH THE COMPLICATION OF DIABETES MELLITUS MEAN VALUES WERE REDUCED (46.00 4.37 M/S FOR THE DEEP PERONEAL NERVE AND 44,36 3.79M/S FOR THE TIBIAL NERVE) WITH A FURTHER STATISTICALLY SIGNIFICANT MCV REDUCTION ONLY FOR THE TIBIAL NERVE. IN 8 PATIENTS NEUROPATHIC FINDINGS WERE RECORDING (EMG OF TIBIALIS ANTERIOR M. AND GASTROCNEMIUS M). IT LOOKS POSSIBLY FROM THE WHOLE STUDY THAT LOW HEAMATOCRITE VALUES ARE RELATED TO THE PATHOGENESIS OF THE NEUROPATHY.","abstract_html":"WE PERFORMED EMG STUDIES IN 53 PATIENTS, TREATED DURING THE PAST FIVE YEARS IN A UNIVERSITY THALASSEMIA UNIT AND IN 29 CONTROLS OF THE SAME AGE RANGE (5 TO 35YEARS). WE STUDIED SEPARATELY SEVEN THALASEMIC PATIENTS WITH THE COMPLICATION OF DIABETES MELLITUS. FROM THE DETAIL CLINICAL EXAMINATION (MEDICAL HISTORY ANDPHYSICAL EXAMINATION) IT WAS FOUND THAT 23% OF THE PATIENTS PRESENTED WITH SENSORY SYMPTOMS (NUMBNESS AND TINGLINGS) IN THE LOWER LIMBS, 17% WITH CRAMPS AND 15% WITH MUSCULAR PAINS. FURTHERMORE 13% OF THE PATIENTS HAD DIMINISHED KNEE JERK AND 19% THE ACHILLES TENDON REFLEX. IN 4% OF PATIENTS THE ACHILLES REFLEX WAS ABSENT. MOTOR CONDUCTION VELOCITIES (MCV) FOR THE DEEP PERONEAL AND THE TIBIAL NERVE WERE REDUCED IN THE PATIENT GROUP COMPARED WITH THE CONTROL GROUP (49.13.37 M/S, SE-0.46 AND 52.2 2.79 M/S, SE=0.51 T-TEST 4.49 FOR THE PERONEAL NERVE AND 48.7 4. 41 M/S, SE=0.56 AND 51.65 3.23 M/S, SE= 0.60 T-TEST 3.58 FOR THE TIBIAL NERVE, RESPECTIVELY). THE GROUP OF THE PATIENTS WITH THE LOWER VALUES OFMCV, WAS THE ONE WHICH SHOWED THE CLINICAL SYMTOMS AND SIGNS, OF PERIPHERAL NEUROPATHY. IN THE GROUP WITH THE COMPLICATION OF DIABETES MELLITUS MEAN VALUES WERE REDUCED (46.00 4.37 M/S FOR THE DEEP PERONEAL NERVE AND 44,36 3.79M/S FOR THE TIBIAL NERVE) WITH A FURTHER STATISTICALLY SIGNIFICANT MCV REDUCTION ONLY FOR THE TIBIAL NERVE. IN 8 PATIENTS NEUROPATHIC FINDINGS WERE RECORDING (EMG OF TIBIALIS ANTERIOR M. AND GASTROCNEMIUS M). IT LOOKS POSSIBLY FROM THE WHOLE STUDY THAT LOW HEAMATOCRITE VALUES ARE RELATED TO THE PATHOGENESIS OF THE NEUROPATHY.","abstract_has_math":false,"creators":["Παπανικολάου, Δημήτρης"],"institution":"University of Patras","degree_name":null,"degree_level":null,"degree_discipline":null,"degree_department":null,"school":null,"contributors":[],"advisors":[],"committee_chairs":[],"committee_members":[],"year":1989,"date_issued":"1989","date_published":"1989","updated_at":"2026-07-24T02:24:43Z","subjects":["ELECTROMYOGRAPHY AND B-THALASSEMIA","ELECTRONEUROGRAPHY AND B-THALASSEMIA","NEUROLOGICAL MANIFESTATIONS OF B-THALASSEMIA","PERIFERAL NEUROPATHY IN B-THALASSEMIA","ΗΛΕΚΤΡΟΜΥΟΓΡΑΦΙΑ ΣΤΗ Β-ΘΑΛΑΣΣΑΙΜΙΑ","ΗΛΕΚΤΡΟΝΕΥΡΟΓΡΑΦΙΑ ΣΤΗ Β-ΘΑΛΑΣΣΑΙΜΙΑ","ΝΕΥΡΟΛΟΓΙΚΕΣ ΕΚΔΗΛΩΣΕΙΣ ΣΤΗ Β-ΘΑΛΑΣΣΑΙΜΙΑ","ΠΕΡΙΦΕΡΙΚΗ ΝΕΥΡΟΠΑΘΕΙΑ ΣΤΗ Β-ΘΑΛΑΣΣΑΙΜΙΑ","Ιατρική και Επιστήμες Υγείας","Κλινική Ιατρική","Medical and Health Sciences","Clinical Medicine"],"languages":["gre"],"rights":[],"rights_urls":[],"identifier_entries":[{"key":"dc:identifier","label":"Identifier","values":["10.12681/eadd/1100"],"render_values":[{"text":"10.12681/eadd/1100","href":"https://doi.org/10.12681/eadd/1100","code":true}]}]},"links":{"outbound_url":"http://hdl.handle.net/10442/hedi/1100","outbound_label":"Handle","outbound_source":"dc:identifier"},"metadata_groups":[{"id":"people","label":"People","entries":[{"key":"dc:creator","label":"Author","values":["Παπανικολάου, Δημήτρης"]}]},{"id":"academic_context","label":"Academic Context","entries":[{"key":"dc:date","label":"Dc Date","values":["1989"]},{"key":"dc:publisher","label":"Institution","values":["University of Patras","Πανεπιστήμιο Πατρών"]},{"key":"dc:type","label":"Dc Type","values":["PhD Thesis"]}]},{"id":"subjects_keywords","label":"Subjects and Keywords","entries":[{"key":"dc:subject","label":"Dc Subject","values":["ELECTROMYOGRAPHY AND B-THALASSEMIA","ELECTRONEUROGRAPHY AND B-THALASSEMIA","NEUROLOGICAL MANIFESTATIONS OF B-THALASSEMIA","PERIFERAL NEUROPATHY IN B-THALASSEMIA","ΗΛΕΚΤΡΟΜΥΟΓΡΑΦΙΑ ΣΤΗ Β-ΘΑΛΑΣΣΑΙΜΙΑ","ΗΛΕΚΤΡΟΝΕΥΡΟΓΡΑΦΙΑ ΣΤΗ Β-ΘΑΛΑΣΣΑΙΜΙΑ","ΝΕΥΡΟΛΟΓΙΚΕΣ ΕΚΔΗΛΩΣΕΙΣ ΣΤΗ Β-ΘΑΛΑΣΣΑΙΜΙΑ","ΠΕΡΙΦΕΡΙΚΗ ΝΕΥΡΟΠΑΘΕΙΑ ΣΤΗ Β-ΘΑΛΑΣΣΑΙΜΙΑ","Ιατρική και Επιστήμες Υγείας","Κλινική Ιατρική","Medical and Health Sciences","Clinical Medicine"]}]},{"id":"language_rights","label":"Language and Rights","entries":[{"key":"dc:language","label":"Dc Language","values":["gre"]}]},{"id":"identifiers","label":"Identifiers","entries":[{"key":"dc:identifier","label":"Identifier","values":["10.12681/eadd/1100","http://hdl.handle.net/10442/hedi/1100"]}]},{"id":"additional","label":"Additional Metadata","entries":[{"key":"dc:description","label":"Description","values":["WE PERFORMED EMG STUDIES IN 53 PATIENTS, TREATED DURING THE PAST FIVE YEARS IN A UNIVERSITY THALASSEMIA UNIT AND IN 29 CONTROLS OF THE SAME AGE RANGE (5 TO 35YEARS). WE STUDIED SEPARATELY SEVEN THALASEMIC PATIENTS WITH THE COMPLICATION OF DIABETES MELLITUS. FROM THE DETAIL CLINICAL EXAMINATION (MEDICAL HISTORY ANDPHYSICAL EXAMINATION) IT WAS FOUND THAT 23% OF THE PATIENTS PRESENTED WITH SENSORY SYMPTOMS (NUMBNESS AND TINGLINGS) IN THE LOWER LIMBS, 17% WITH CRAMPS AND 15% WITH MUSCULAR PAINS. FURTHERMORE 13% OF THE PATIENTS HAD DIMINISHED KNEE JERK AND 19% THE ACHILLES TENDON REFLEX. IN 4% OF PATIENTS THE ACHILLES REFLEX WAS ABSENT. MOTOR CONDUCTION VELOCITIES (MCV) FOR THE DEEP PERONEAL AND THE TIBIAL NERVE WERE REDUCED IN THE PATIENT GROUP COMPARED WITH THE CONTROL GROUP (49.13.37 M/S, SE-0.46 AND 52.2 2.79 M/S, SE=0.51 T-TEST 4.49 FOR THE PERONEAL NERVE AND 48.7 4. 41 M/S, SE=0.56 AND 51.65 3.23 M/S, SE= 0.60 T-TEST 3.58 FOR THE TIBIAL NERVE, RESPECTIVELY). THE GROUP OF THE PATIENTS WITH THE LOWER VALUES OFMCV, WAS THE ONE WHICH SHOWED THE CLINICAL SYMTOMS AND SIGNS, OF PERIPHERAL NEUROPATHY. IN THE GROUP WITH THE COMPLICATION OF DIABETES MELLITUS MEAN VALUES WERE REDUCED (46.00 4.37 M/S FOR THE DEEP PERONEAL NERVE AND 44,36 3.79M/S FOR THE TIBIAL NERVE) WITH A FURTHER STATISTICALLY SIGNIFICANT MCV REDUCTION ONLY FOR THE TIBIAL NERVE. IN 8 PATIENTS NEUROPATHIC FINDINGS WERE RECORDING (EMG OF TIBIALIS ANTERIOR M. AND GASTROCNEMIUS M). IT LOOKS POSSIBLY FROM THE WHOLE STUDY THAT LOW HEAMATOCRITE VALUES ARE RELATED TO THE PATHOGENESIS OF THE NEUROPATHY.","ΜΕΛΕΤΗΣΑΜΕ ΚΛΙΝΙΚΑ ΚΑΙ ΗΛΕΚΤΡΟΦΥΣΙΟΛΟΓΙΚΑ 53 ΘΑΛΑΣΣΑΙΜΙΚΟΥΣ ΑΣΘΕΝΕΙΣ ΚΑΙ ΕΠΙΠΛΕΟΝ ΞΕΧΩΡΙΣΤΑ 7 ΘΑΛΑΣΣΑΙΜΙΚΟΥΣ ΑΣΘΕΝΕΙΣ ΜΕ ΤΗΝ ΕΠΙΠΛΟΚΗ ΤΟΥ ΣΑΚΧΑΡΩΔΟΥΣ ΔΙΑΒΗΤΟΥ. ΑΠΟ ΤΗΝ ΛΕΠΤΟΜΕΡΗ ΚΛΙΝΙΚΗ ΜΕΛΕΤΗ (ΙΣΤΟΡΙΚΟ ΚΑΙ ΑΝΤΙΚΕΙΜΕΝΙΚΗ ΝΕΥΡΟΛΟΓΙΚΗ ΕΞΕΤΑΣΗ) ΔΙΑΠΙΣΤΘΩΘΗΚΕ ΟΤΙ 23% ΤΩΝ ΑΣΘΕΝΩΝ ΠΑΡΑΠΟΝΕΙΤΟ ΓΙΑ ΑΙΜΩΔΙΕΣ ΚΑΙ ΑΙΣΘΗΜΑ ΝΥΓΜΩΝ ΣΤΑ ΚΑΤΑ ΑΚΡΑ, 17% ΓΙΑ ΜΥΙΚΕΣ ΚΡΑΜΠΕΣ ΚΑΙ 15% ΓΙΑ ΜΥΑΛΓΙΕΣ. ΑΚΟΜΗ 13% ΠΑΡΟΥΣΙΑΖΑΝ ΜΕΙΩΜΕΝΟ ΕΠΙΓΟΝΑΤΙΔΙΚΟ ΑΝΤΑΝΑΚΛΑΣΤΙΚΟ, 19% ΜΕΙΩΜΕΝΟ ΚΑΙ 4% ΚΑΤΑΡΓΗΜΕΝΟ ΑΧΙΛΛΕΙΟ ΑΝΤΑΝΑΚΛΑΣΤΙΚΟ. Η ΚΙΝΗΤΙΚΗ ΤΑΧΥΤΗΤΑ ΑΓΩΓΗΣ ΓΙΑ ΤΟ ΠΕΡΙΝΙΑΙΟ ΚΑΙ ΤΟ ΚΝΗΜΙΑΙΟ ΝΕΥΡΟ, ΗΤΑΝ ΜΕΙΩΜΕΝΗ ΣΤΗΝ ΟΜΑΔΑ ΤΩΝ ΑΣΘΕΝΩΝ ΣΕ ΣΧΕΣΗ ΜΕ ΤΗΝ ΟΜΑΔΑ ΤΩΝ ΜΑΡΤΥΡΩΝ (49.1 3.37 M/S, SE=0.46 ΚΑΙ 52.2 2.79 M/S SE=0.51, T-TEST=4.49 ΓΙΑ ΤΟ ΠΕΡΟΝΙΑΙΟ ΝΕΥΡΟ ΚΑΙ 48.7 4.14 M/S SE=0.56 ΚΑΙ 51.65 3.23 M/S SE=0.60,T-TEST=3.58 ΓΙΑ ΤΟ ΚΝΗΜΙΑΙΟ ΝΕΥΡΟ, ΑΝΤΙΣΤΟΙΧΩΣ). ΟΙ ΑΣΘΕΝΕΙΣ ΠΟΥ ΕΙΧΑΝ ΤΙΣ ΧΑΜΗΛΟΤΕΡΕΣ ΤΙΜΕΣ ΚΙΝΗΤΙΚΗΣ ΤΑΧΥΤΗΤΑΣ ΑΓΩΓΗΣ ΕΙΧΑΝ ΚΑΙ ΕΥΡΗΜΑΤΑ ΑΠΟ ΤΗΝ ΚΛΙΝΙΚΗ ΕΞΕΤΑΣΗ. ΣΤΗΝ ΟΜΑΔΑ ΤΩΝ ΘΑΛΑΣΣΑΙΜΙΚΩΝ ΑΣΘΕΝΩΝ ΜΕ ΤΗΝ ΕΠΙΠΛΟΚΗ ΤΟΥ ΣΑΚΧΑΡΩΔΟΥΣ ΔΙΑΒΗΤΟΥ Η ΜΕΣΗ ΤΙΜΗ ΤΗΣ ΚΙΝΗΤΙΚΗΣ ΤΑΧΥΤΗΤΑΣ ΑΓΩΓΗΣ ΗΤΑΝ ΜΕΙΩΜΕΝΗ (46.00 4.37 M/S ΓΙΑ ΤΟ ΠΕΡΟΝΙΑΙΟ ΝΕΥΡΟ ΚΑΙ 44.36 3 .79 M/S ΓΙΑ ΤΟ ΚΝΗΜΙΑΙΟ ΝΕΥΡΟ). ΕΠΙΣΗΣ ΔΙΑΠΙΣΤΩΘΗΚΕ ΜΙΑ ΣΤΑΤΙΣΤΙΚΑ ΣΗΜΑΝΤΙΚΗ ΔΙΑΦΟΡΑ ΤΗΣ ΚΙΝΗΤΙΚΗΣ ΤΑΧΥΤΗΤΑΣ ΑΓΩΓΗΣ (ΜΕΙΩΣΗ) ΤΗΝ ΚΝΗΜΙΑΙΟΥΝΕΥΡΟΥ ΣΤΗΝ ΟΜΑΔΑ ΘΑΛΑΣΣΑΙΜΙΚΩΝ ΑΣΘΕΝΩΝ ΜΕ ΣΑΚΧΑΡΩΔΗ ΔΙΑΒΗΤΗ ΣΕ ΣΧΕΣΗ ΜΕ ΤΟΥΣ ΘΑΛΑΣΣΑΙΜΙΚΟΥΣ ΑΣΘΕΝΕΙΣ ΠΟΥ ΜΕΛΕΤΗΣΑΜΕ (53 ΑΣΘΕΝΕΙΣ). ΣΕ 8 ΑΣΘΕΝΕΙΣ ΣΤΟΝ ΠΡΟΣΘΙΟ ΚΝΗΜΙΑΙΟ ΚΑΙ ΓΑΣΤΡΟΚΝΗΜΙΟ ΜΥ ΒΡΕΘΗΚΑΝ ΝΕΥΡΟΠΑΘΗΤΙΚΑ ΣΤΟΙΧΕΙΑ ΑΠΟ ΤΗΝ ΗΛΕΚΤΡΟΜΥΟΓΡΑΦΙΚΗ ΕΞΕΤΑΣΗ. ΑΠΟ ΤΑ ΣΤΟΙΧΕΙΑ ΤΗΣ ΜΕΛΕΤΗΣ ΜΑΣ ΦΑΙΝΕΤΑΙ ΠΕΡΙΣΣΟΤΕΡΟ ΠΙΘΑΝΟΝΟΤΙ Ο ΠΑΘΟΓΕΝΕΤΙΚΟΣ ΜΗΧΑΝΙΣΜΟΣ ΣΥΝΔΕΕΤΑΙ ΜΕ ΤΟΝ ΧΑΜΗΛΟ ΑΙΜΑΤΟΚΡΙΤΗ."]},{"key":"dc:title","label":"Title","values":["ΝΕΥΡΟΦΥΣΙΟΛΟΓΙΚΗ ΜΕΛΕΤΗ ΤΟΥ ΠΕΡΙΦΕΡΙΚΟΥ ΝΕΥΡΙΚΟΥ ΚΑΙ ΜΥΙΚΟΥ ΣΥΣΤΗΜΑΤΟΣ ΣΕ ΑΣΘΕΝΕΙΣ ΜΕ ΟΜΟΖΥΓΟ Β-ΘΑΛΑΣΣΑΙΜΙΑ","A NEUROPHYSIOLOGICAL STUDY OF THE PERIFERAL NERVOUS AND MUSCULAR SYSTEM IN B-THALASSEMIA"]}]}],"canonical_facts":{"dc:creator":["Παπανικολάου, Δημήτρης"],"dc:date":["1989"],"dc:description":["WE PERFORMED EMG STUDIES IN 53 PATIENTS, TREATED DURING THE PAST FIVE YEARS IN A UNIVERSITY THALASSEMIA UNIT AND IN 29 CONTROLS OF THE SAME AGE RANGE (5 TO 35YEARS). WE STUDIED SEPARATELY SEVEN THALASEMIC PATIENTS WITH THE COMPLICATION OF DIABETES MELLITUS. FROM THE DETAIL CLINICAL EXAMINATION (MEDICAL HISTORY ANDPHYSICAL EXAMINATION) IT WAS FOUND THAT 23% OF THE PATIENTS PRESENTED WITH SENSORY SYMPTOMS (NUMBNESS AND TINGLINGS) IN THE LOWER LIMBS, 17% WITH CRAMPS AND 15% WITH MUSCULAR PAINS. FURTHERMORE 13% OF THE PATIENTS HAD DIMINISHED KNEE JERK AND 19% THE ACHILLES TENDON REFLEX. IN 4% OF PATIENTS THE ACHILLES REFLEX WAS ABSENT. MOTOR CONDUCTION VELOCITIES (MCV) FOR THE DEEP PERONEAL AND THE TIBIAL NERVE WERE REDUCED IN THE PATIENT GROUP COMPARED WITH THE CONTROL GROUP (49.13.37 M/S, SE-0.46 AND 52.2 2.79 M/S, SE=0.51 T-TEST 4.49 FOR THE PERONEAL NERVE AND 48.7 4. 41 M/S, SE=0.56 AND 51.65 3.23 M/S, SE= 0.60 T-TEST 3.58 FOR THE TIBIAL NERVE, RESPECTIVELY). THE GROUP OF THE PATIENTS WITH THE LOWER VALUES OFMCV, WAS THE ONE WHICH SHOWED THE CLINICAL SYMTOMS AND SIGNS, OF PERIPHERAL NEUROPATHY. IN THE GROUP WITH THE COMPLICATION OF DIABETES MELLITUS MEAN VALUES WERE REDUCED (46.00 4.37 M/S FOR THE DEEP PERONEAL NERVE AND 44,36 3.79M/S FOR THE TIBIAL NERVE) WITH A FURTHER STATISTICALLY SIGNIFICANT MCV REDUCTION ONLY FOR THE TIBIAL NERVE. IN 8 PATIENTS NEUROPATHIC FINDINGS WERE RECORDING (EMG OF TIBIALIS ANTERIOR M. AND GASTROCNEMIUS M). IT LOOKS POSSIBLY FROM THE WHOLE STUDY THAT LOW HEAMATOCRITE VALUES ARE RELATED TO THE PATHOGENESIS OF THE NEUROPATHY.","ΜΕΛΕΤΗΣΑΜΕ ΚΛΙΝΙΚΑ ΚΑΙ ΗΛΕΚΤΡΟΦΥΣΙΟΛΟΓΙΚΑ 53 ΘΑΛΑΣΣΑΙΜΙΚΟΥΣ ΑΣΘΕΝΕΙΣ ΚΑΙ ΕΠΙΠΛΕΟΝ ΞΕΧΩΡΙΣΤΑ 7 ΘΑΛΑΣΣΑΙΜΙΚΟΥΣ ΑΣΘΕΝΕΙΣ ΜΕ ΤΗΝ ΕΠΙΠΛΟΚΗ ΤΟΥ ΣΑΚΧΑΡΩΔΟΥΣ ΔΙΑΒΗΤΟΥ. ΑΠΟ ΤΗΝ ΛΕΠΤΟΜΕΡΗ ΚΛΙΝΙΚΗ ΜΕΛΕΤΗ (ΙΣΤΟΡΙΚΟ ΚΑΙ ΑΝΤΙΚΕΙΜΕΝΙΚΗ ΝΕΥΡΟΛΟΓΙΚΗ ΕΞΕΤΑΣΗ) ΔΙΑΠΙΣΤΘΩΘΗΚΕ ΟΤΙ 23% ΤΩΝ ΑΣΘΕΝΩΝ ΠΑΡΑΠΟΝΕΙΤΟ ΓΙΑ ΑΙΜΩΔΙΕΣ ΚΑΙ ΑΙΣΘΗΜΑ ΝΥΓΜΩΝ ΣΤΑ ΚΑΤΑ ΑΚΡΑ, 17% ΓΙΑ ΜΥΙΚΕΣ ΚΡΑΜΠΕΣ ΚΑΙ 15% ΓΙΑ ΜΥΑΛΓΙΕΣ. ΑΚΟΜΗ 13% ΠΑΡΟΥΣΙΑΖΑΝ ΜΕΙΩΜΕΝΟ ΕΠΙΓΟΝΑΤΙΔΙΚΟ ΑΝΤΑΝΑΚΛΑΣΤΙΚΟ, 19% ΜΕΙΩΜΕΝΟ ΚΑΙ 4% ΚΑΤΑΡΓΗΜΕΝΟ ΑΧΙΛΛΕΙΟ ΑΝΤΑΝΑΚΛΑΣΤΙΚΟ. Η ΚΙΝΗΤΙΚΗ ΤΑΧΥΤΗΤΑ ΑΓΩΓΗΣ ΓΙΑ ΤΟ ΠΕΡΙΝΙΑΙΟ ΚΑΙ ΤΟ ΚΝΗΜΙΑΙΟ ΝΕΥΡΟ, ΗΤΑΝ ΜΕΙΩΜΕΝΗ ΣΤΗΝ ΟΜΑΔΑ ΤΩΝ ΑΣΘΕΝΩΝ ΣΕ ΣΧΕΣΗ ΜΕ ΤΗΝ ΟΜΑΔΑ ΤΩΝ ΜΑΡΤΥΡΩΝ (49.1 3.37 M/S, SE=0.46 ΚΑΙ 52.2 2.79 M/S SE=0.51, T-TEST=4.49 ΓΙΑ ΤΟ ΠΕΡΟΝΙΑΙΟ ΝΕΥΡΟ ΚΑΙ 48.7 4.14 M/S SE=0.56 ΚΑΙ 51.65 3.23 M/S SE=0.60,T-TEST=3.58 ΓΙΑ ΤΟ ΚΝΗΜΙΑΙΟ ΝΕΥΡΟ, ΑΝΤΙΣΤΟΙΧΩΣ). ΟΙ ΑΣΘΕΝΕΙΣ ΠΟΥ ΕΙΧΑΝ ΤΙΣ ΧΑΜΗΛΟΤΕΡΕΣ ΤΙΜΕΣ ΚΙΝΗΤΙΚΗΣ ΤΑΧΥΤΗΤΑΣ ΑΓΩΓΗΣ ΕΙΧΑΝ ΚΑΙ ΕΥΡΗΜΑΤΑ ΑΠΟ ΤΗΝ ΚΛΙΝΙΚΗ ΕΞΕΤΑΣΗ. ΣΤΗΝ ΟΜΑΔΑ ΤΩΝ ΘΑΛΑΣΣΑΙΜΙΚΩΝ ΑΣΘΕΝΩΝ ΜΕ ΤΗΝ ΕΠΙΠΛΟΚΗ ΤΟΥ ΣΑΚΧΑΡΩΔΟΥΣ ΔΙΑΒΗΤΟΥ Η ΜΕΣΗ ΤΙΜΗ ΤΗΣ ΚΙΝΗΤΙΚΗΣ ΤΑΧΥΤΗΤΑΣ ΑΓΩΓΗΣ ΗΤΑΝ ΜΕΙΩΜΕΝΗ (46.00 4.37 M/S ΓΙΑ ΤΟ ΠΕΡΟΝΙΑΙΟ ΝΕΥΡΟ ΚΑΙ 44.36 3 .79 M/S ΓΙΑ ΤΟ ΚΝΗΜΙΑΙΟ ΝΕΥΡΟ). ΕΠΙΣΗΣ ΔΙΑΠΙΣΤΩΘΗΚΕ ΜΙΑ ΣΤΑΤΙΣΤΙΚΑ ΣΗΜΑΝΤΙΚΗ ΔΙΑΦΟΡΑ ΤΗΣ ΚΙΝΗΤΙΚΗΣ ΤΑΧΥΤΗΤΑΣ ΑΓΩΓΗΣ (ΜΕΙΩΣΗ) ΤΗΝ ΚΝΗΜΙΑΙΟΥΝΕΥΡΟΥ ΣΤΗΝ ΟΜΑΔΑ ΘΑΛΑΣΣΑΙΜΙΚΩΝ ΑΣΘΕΝΩΝ ΜΕ ΣΑΚΧΑΡΩΔΗ ΔΙΑΒΗΤΗ ΣΕ ΣΧΕΣΗ ΜΕ ΤΟΥΣ ΘΑΛΑΣΣΑΙΜΙΚΟΥΣ ΑΣΘΕΝΕΙΣ ΠΟΥ ΜΕΛΕΤΗΣΑΜΕ (53 ΑΣΘΕΝΕΙΣ). ΣΕ 8 ΑΣΘΕΝΕΙΣ ΣΤΟΝ ΠΡΟΣΘΙΟ ΚΝΗΜΙΑΙΟ ΚΑΙ ΓΑΣΤΡΟΚΝΗΜΙΟ ΜΥ ΒΡΕΘΗΚΑΝ ΝΕΥΡΟΠΑΘΗΤΙΚΑ ΣΤΟΙΧΕΙΑ ΑΠΟ ΤΗΝ ΗΛΕΚΤΡΟΜΥΟΓΡΑΦΙΚΗ ΕΞΕΤΑΣΗ. ΑΠΟ ΤΑ ΣΤΟΙΧΕΙΑ ΤΗΣ ΜΕΛΕΤΗΣ ΜΑΣ ΦΑΙΝΕΤΑΙ ΠΕΡΙΣΣΟΤΕΡΟ ΠΙΘΑΝΟΝΟΤΙ Ο ΠΑΘΟΓΕΝΕΤΙΚΟΣ ΜΗΧΑΝΙΣΜΟΣ ΣΥΝΔΕΕΤΑΙ ΜΕ ΤΟΝ ΧΑΜΗΛΟ ΑΙΜΑΤΟΚΡΙΤΗ."],"dc:identifier":["10.12681/eadd/1100","http://hdl.handle.net/10442/hedi/1100"],"dc:language":["gre"],"dc:publisher":["University of Patras","Πανεπιστήμιο Πατρών"],"dc:subject":["ELECTROMYOGRAPHY AND B-THALASSEMIA","ELECTRONEUROGRAPHY AND B-THALASSEMIA","NEUROLOGICAL MANIFESTATIONS OF B-THALASSEMIA","PERIFERAL NEUROPATHY IN B-THALASSEMIA","ΗΛΕΚΤΡΟΜΥΟΓΡΑΦΙΑ ΣΤΗ Β-ΘΑΛΑΣΣΑΙΜΙΑ","ΗΛΕΚΤΡΟΝΕΥΡΟΓΡΑΦΙΑ ΣΤΗ Β-ΘΑΛΑΣΣΑΙΜΙΑ","ΝΕΥΡΟΛΟΓΙΚΕΣ ΕΚΔΗΛΩΣΕΙΣ ΣΤΗ Β-ΘΑΛΑΣΣΑΙΜΙΑ","ΠΕΡΙΦΕΡΙΚΗ ΝΕΥΡΟΠΑΘΕΙΑ ΣΤΗ Β-ΘΑΛΑΣΣΑΙΜΙΑ","Ιατρική και Επιστήμες Υγείας","Κλινική Ιατρική","Medical and Health Sciences","Clinical Medicine"],"dc:title":["ΝΕΥΡΟΦΥΣΙΟΛΟΓΙΚΗ ΜΕΛΕΤΗ ΤΟΥ ΠΕΡΙΦΕΡΙΚΟΥ ΝΕΥΡΙΚΟΥ ΚΑΙ ΜΥΙΚΟΥ ΣΥΣΤΗΜΑΤΟΣ ΣΕ ΑΣΘΕΝΕΙΣ ΜΕ ΟΜΟΖΥΓΟ Β-ΘΑΛΑΣΣΑΙΜΙΑ","A NEUROPHYSIOLOGICAL STUDY OF THE PERIFERAL NERVOUS AND MUSCULAR SYSTEM IN B-THALASSEMIA"],"dc:type":["PhD Thesis"]},"updated_at":"2026-07-24T02:24:43Z"}