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Showing 1 to 7 of 7 for “"sensory ganglia"”.

  1. The morphology, neurochemistry, and consequences of sympathosensory plexuses

    … devoid of sympathetic fibers, such as the sensory ganglia. When postganglionic sympathetic axons grow into the environment of sensory ganglia, they can converge and wrap around a subset of somata (i.e., cell bodies) belonging to primary sensory neurons. This phenomenon, referred to as …

    queens Repository record for The morphology, neurochemistry, and consequences of sympathosensory plexuses (opens in a new tab)

  2. Molecular analysis of herpes simplex virus type 1 latency in experimentally infected mice / Barry Slobedman.

    … nucleic acid sequences in latently infected sensory ganglia, experiments are undertaken using a mouse model that makes novel use of the segmental sensory innervation of flank skin.

    adelaide Repository record for Molecular analysis of herpes simplex virus type 1 latency in experimentally infected mice / Barry Slobedman. (opens in a new tab)

  3. Transcriptional analysis of the role of CD8+ T lymphocytes in acute neural herpes simplex virus infection / David C. Tscharke.

    … with CD8+ T lymphocyte activity in HSV infected sensory ganglia. The role of CD8+ T cells in cytokine responses to ganglionic HSV infection is investigated, with particular reference to the Th1/Th2 paradigm and a known anti-viral mediator, IFN-[gamma]. A non-directed method of mRNA analysis is …

    adelaide Repository record for Transcriptional analysis of the role of CD8+ T lymphocytes in acute neural herpes simplex virus infection / David C. Tscharke. (opens in a new tab)

  4. Effector CD4+ T lymphocyte resolution of acute HSV infection at genital and neuronal sites, and the manipulation of CD4+ T cell responses via TLR ligand-induced proinflammatory cytokine milieus

    … important for clearance of infectious HSV from sensory ganglia. We present evidence of CD4+ T-cell-mediated clearance of infectious HSV-1 from neural tissues. In immunocompetent mice, HSV-specific CD4+ T cells were present in sensory ganglia and spinal cords coincident with HSV-1 clearance and …

    utmb Repository record for Effector CD4+ T lymphocyte resolution of acute HSV infection at genital and neuronal sites, and the manipulation of CD4+ T cell responses via TLR ligand-induced proinflammatory cytokine milieus (opens in a new tab)

  5. Functional Analysis of SOX11 and NF1 in Sensory Neuron Development and Plasticity

    Development of sensory neurons is controlled by both cell-extrinsic and cell-intrinsic factors. The transcription factor Sox11 is abundantly expressed in embryonic sensory neurons. In the first part of this thesis, by using a Sox11-/- mouse model, I show that the loss of Sox11 results in increased …

    utswmed Repository record for Functional Analysis of SOX11 and NF1 in Sensory Neuron Development and Plasticity (opens in a new tab)

  6. Topological organization of the trigeminal system in the lamprey and restoration following axonal regeneration

    … locomotor behavior recovers in a few weeks. The sensory and motor areas of the vertebrate nervous system often exhibit a topological or somatotopic organization, presumably to facilitate efficient transmission and processing of information. Preliminary results suggested the existence of a …

    missouri Repository record for Topological organization of the trigeminal system in the lamprey and restoration following axonal regeneration (opens in a new tab)

  7. Molecular basis oh herpes simplex virus entry into the cell and retargeting of the viral tropism for the design of oncolytic herpesviruses

    … to the nerve endings and estabilishes latency in sensory ganglia, from where it may, or may not reactivate. Diseases caused by virus reactivation include mild diseases such as muco-cutaneous lesions, and more severe, and even life-threatening encephalitis, or systemic infections affecting diverse …

    bologna Repository record for Molecular basis oh herpes simplex virus entry into the cell and retargeting of the viral tropism for the design of oncolytic herpesviruses (opens in a new tab)