Global ETD Search

Search theses and dissertations gathered from participating repositories worldwide. Every result links back to the library that holds it. No account is needed.

Results

Showing 1 to 17 of 17 for “"primary cortical neurons"”.

  1. An in vitro study to characterize a new automated high throughput neuronal stretch injury system

    … investigation, a new methodology for culturing primary cortical neurons of rat origin was established. Viable cell cultures developed for the first time in NJIT laboratory were successfully injured using the new NJIT device. The data recorded here may allow us to understand better the complex …

    njit Repository record for An in vitro study to characterize a new automated high throughput neuronal stretch injury system (opens in a new tab)

  2. Astrocytes Rescue Neuronal Health After Cisplatin Treatment Through Mitochondrial Transfer.

    … that astrocytes transfer healthy mitochondria to neurons after cisplatin treatment to restore neuronal health.</p> <p>We used an <em>in vitro</em> system in which astrocytes with Mito-mCherry-labeled mitochondria were co-cultured with primary cortical neurons or neuronal stem cells damaged by …

    uthsc Repository record for Astrocytes Rescue Neuronal Health After Cisplatin Treatment Through Mitochondrial Transfer. (opens in a new tab)

  3. Neuroprotection of AG490 in Oxidative Stress-induced Neuronal Death

    … stress-induced cytotoxicity in cultured primary cortical neurons. AG490 significantly protected neuronal cell death against various oxidative stimuli, such as hydrogen peroxide (H2O2), L-buthionin-S,R-sulfoxamine (BSO), N-methyl-D-aspartic acid (NMDA), and arachidonic acid (AA). …

    ajou Repository record for Neuroprotection of AG490 in Oxidative Stress-induced Neuronal Death (opens in a new tab)

  4. Targeted Gene Therapy Approaches for Huntington's Disease: Advancing Treatment through Bioengineered Self-Assembling Peptide Hydrogels and BDNF-Encoded Adeno-Associated Viruses, Applied In Vitro and In Vivo

    … protein. It mainly enhances the loss of striatal neurons and primary cortical neurons within the forebrain. Gene therapy has provided promising targeted therapeutic strategies to treat various neurodegenerative disorders such as HD, where the genomic DNA of a cell can be rewritten, reengineered, …

    aus-cath Repository record for Targeted Gene Therapy Approaches for Huntington's Disease: Advancing Treatment through Bioengineered Self-Assembling Peptide Hydrogels and BDNF-Encoded Adeno-Associated Viruses, Applied In Vitro and In Vivo (opens in a new tab)

  5. Targeted Gene Therapy Approaches for Huntington's Disease: Advancing Treatment through Bioengineered Self-Assembling Peptide Hydrogels and BDNF-Encoded Adeno-Associated Viruses, Applied In Vitro and In Vivo

    … protein. It mainly enhances the loss of striatal neurons and primary cortical neurons within the forebrain. Gene therapy has provided promising targeted therapeutic strategies to treat various neurodegenerative disorders such as HD, where the genomic DNA of a cell can be rewritten, reengineered, …

    anu Repository record for Targeted Gene Therapy Approaches for Huntington's Disease: Advancing Treatment through Bioengineered Self-Assembling Peptide Hydrogels and BDNF-Encoded Adeno-Associated Viruses, Applied In Vitro and In Vivo (opens in a new tab)

  6. GPR50, a potential factor involved in psychiatric disorders interacts with Alzheimer’s disease-related protein β- secretase (BACE1)

    … in HEK293 cells, and at the neurites in mouse primary neuronal cells. Subcellular fractionation of adult mouse brain revealed that endogenous Gpr50 and Bace1 were co-fractionated in the presynaptic vesicles. Secondly, I showed that, in contrast to HEK293 cells, GPR50 overexpression had no …

    edinburgh Repository record for GPR50, a potential factor involved in psychiatric disorders interacts with Alzheimer’s disease-related protein β- secretase (BACE1) (opens in a new tab)

  7. Analyzing A-series gangliosides in neurons following exposure to glutamate

    Neurons within different brain regions have varying levels of vulnerability to external stress and therefore respond differently to injury. A potential reason to explain this may lie within a key lipid class of the cell’s plasma membrane called gangliosides. These glycosphingolipid species have …

    uwo Repository record for Analyzing A-series gangliosides in neurons following exposure to glutamate (opens in a new tab)

  8. Neuronal Protection by a Novel C-terminal Hsp90 Modulator

    … non-toxic Hsp90 modulator, KU-32, which protects primary cortical neurons against Aβ1-42, a toxic peptide found in AD brain. Pharmacokinetic studies indicated that KU-32 is blood brain barrier permeable and orally bioavailable. Using two AD mouse models, JNPL3 and rTg4510, which both contain the …

    ku Repository record for Neuronal Protection by a Novel C-terminal Hsp90 Modulator (opens in a new tab)

  9. Regulation of mGluR7 trafficking by SUMOylatio in neurons

    … of full-length mGluR7 in mammalian cells and neurons. Here we have explored whether mGluR7 is a target of SUMOylation. Using biochemical approaches coupled with confocal imaging, we find that mGluR7 at Lys889, the sole SUMOylation site on the C-terminus of mGluR7 is a target of SUMO …

    ajou Repository record for Regulation of mGluR7 trafficking by SUMOylatio in neurons (opens in a new tab)

  10. Interplay of Brain Derived Neurotrophic Factor and Protein Kinase A in Modulating Mitochondrial Structure and Function in Brain Homeostasis and Disease

    Abstract:Neurons are postmitotic cells that contain elongated specialized structures that emanate from their cell bodies termed neurites (axons and dendrites). In order to form neuronal networks, neurons rely on continuous neurotrophic support mediated by Brain-Derived Neurotrophic Factor (BDNF). …

    unr Repository record for Interplay of Brain Derived Neurotrophic Factor and Protein Kinase A in Modulating Mitochondrial Structure and Function in Brain Homeostasis and Disease (opens in a new tab)

  11. Investigation into the destructive and adaptive responses of neural cells to stress

    … unit is essential to the longevity of neurons. Conversely, loss of homeostasis, particularly of Ca2+ levels, of redox balance and of ATP, contribute to neuronal loss and dysfunction in many neurodegenerative and neurological disorders. This thesis is centred on better understanding the …

    edinburgh Repository record for Investigation into the destructive and adaptive responses of neural cells to stress (opens in a new tab)

  12. Examinations into the Calcium Hypothesis of Alzheimer's Disease

    … level rescues exaggerated [Ca2+]i signaling in primary cortical neurons and hippocampal slices from mutant PS1 expressing mice. We then determined the contribution of exaggerated [Ca2+]i signaling to AD pathogenesis. Using a combination of genetic, biochemical, electrophysiological and …

    penn Repository record for Examinations into the Calcium Hypothesis of Alzheimer's Disease (opens in a new tab)

  13. Utilizing Toxoplasma gondii To Understand Neuron-Specific Neuroinflammation

    … persist for the life of the host by hiding in neurons in the Central Nervous System (CNS). It was thought that neurons permitted this chronic infection due to a lack of innate immune capabilities including decreased expression of cytokine and immune cell receptors, as well as an inability to …

    arizona-thes Repository record for Utilizing Toxoplasma gondii To Understand Neuron-Specific Neuroinflammation (opens in a new tab)

  14. Neuroprotektive Effekte von Atorvastatin bei Glutamat-induzierter Exzitotoxizität in primären kortikalen Neuronen

    Atorvastatin gehört zu einer Gruppe lipidsenkender Medikamente, die über eine kompetitive Hemmung des Schlüsselenzyms der Cholesterin-Biosynthese, der -HMG-CoA-Reduktase (-Hydroxymethylglutaryl-Coenzym A-Reduktase) wirken. Sie sind daher zur Therapie der Hypercholesterinämie (Stein et al. 2002) …

    fu-berlin Repository record for Neuroprotektive Effekte von Atorvastatin bei Glutamat-induzierter Exzitotoxizität in primären kortikalen Neuronen (opens in a new tab)

  15. Promoting Central Nervous System Regeneration by Targeting Intracellular Pathways

    Adult central nervous system neurons regenerate poorly after injury. One reason for this regenerative failure is that a developmental change occurs where essential growth-molecules become excluded from axons with maturation. In this thesis, two strategies were employed in order to improve axon …

    cambridge Repository record for Promoting Central Nervous System Regeneration by Targeting Intracellular Pathways (opens in a new tab)

  16. Isopeptide and Ester Bond Ubiquitination Regulate Degradation of the Human Dopamine Receptor 4

    … cognition and circadian rhythms. The hD4R is the primary dopaminergic receptor for the atypical anti-psychotic drug clozapine, which is used to treat schizophrenia and other cognitive disorders. The hD4R gene is unique among the superfamily of GPCR-encoding genes because within the human …

    rockefeller Repository record for Isopeptide and Ester Bond Ubiquitination Regulate Degradation of the Human Dopamine Receptor 4 (opens in a new tab)

  17. Structural Properties of α-Synuclein in Functional and Pathological Contexts

    … known as Lewy bodies that form inside the neurons of patients suffering from these conditions. A number of missense mutations, as well as duplications and triplications of the gene encoding αS have also been associated with familial forms of early onset PD. Despite the association between …

    cambridge Repository record for Structural Properties of α-Synuclein in Functional and Pathological Contexts (opens in a new tab)