Global ETD Search
Search theses and dissertations gathered from participating repositories worldwide. Every result links back to the library that holds it. No account is needed.
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Showing 1 to 5 of 5 for “"hypertrophic scar"”.
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Hypertrophic scar therapy : pressure-induced remodelling and its determinents
Hypertrophic scars are cosmetically unattractive products of abnormal wound healing and, if they occur over flexor aspects of joints, considerable functional impairment often results. Pressure, as a therapy for hypertrophic- scarring has considerable attraction since it is effective and …
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Mast Cell Influence in the Progression of Post-burn Hypertrophic Scar Pathophysiology
Painful, motion-limiting hypertrophic scars (HTS) form after protracted wound healing in patients with severe full-thickness burns and pose difficult treatment challenges. Despite substantial reduction in postburn mortality, long-term outcomes of HTS are the most serious therapeutic challenge …
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Hypertrophic scar tissue : its microstructure and mechanical properties and the effects of pressure therapy
The formation of hypertrophic scar tissue during the process of wound healing results in severe disfiguring and disabling consequences for the patient. This thesis presents the detailed micro-structure of hypertrophic scar tissue, as viewed with the S.E.M. and discusses the inter-relationship …
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The effect of low level light therapy on wound healing: in vitro study with normal and keloid fibroblast
… light. LLLT is used in clinically to preventing hypertrophic scar or keloid and stimulating wound healing, while the mechanism of their effects remains unclear. Therefore, we investigated the effect of 415 nm, 632 nm, and 830 nm light emitting diodes (LED) to wound healing, especially in normal …
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Fibroblasts: Key Cells in Inflammation and Fibrosis
… the myofibroblast phenotype in cells taken from hypertrophic scars of burn patients. In conclusion, my work suggests that fibroblast-RelA is not necessary for myofibroblast transdifferentiation but it plays pivotal role in Ang II mediated vascular inflammation and AAA formation.