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Showing 1 to 8 of 8 for “"hotspot mutation"”.

  1. Persistent Oxidative Damage Accounts for the Origin of Mutations Observed in Uterine Pathology

    … gene. The type I endometrial cancer PTEN hotspot mutation involves a cytosine to guanine transversion. The mechanism for C to G transversions in endometrial tissue has not been previously explained. For the first time, we show that the CpG dinucleotide of codon 130 is methylated, which …

    utmb Repository record for Persistent Oxidative Damage Accounts for the Origin of Mutations Observed in Uterine Pathology (opens in a new tab)

  2. Cooperative Genomic Events Driving Mutant TP53-Driven DCIS Progression

    … and breast cancer suggest that <em>TP53</em> mutations occur early in DCIS, suggesting a critical role for mutant <em>TP53</em> in driving disease progression. Using a somatic mouse model of <em>p53<sup>R245W</sup></em> induced breast cancer (equivalent to the <em>TP53<sup>R248W</sup></em> …

    uthsc Repository record for Cooperative Genomic Events Driving Mutant TP53-Driven DCIS Progression (opens in a new tab)

  3. Omics Approaches to Uncover Germline and Somatic Variation Underlying Inherited Sarcomagenesis

    … a high incidence of sarcomas. LFS is caused by mutations in the tumor suppressor gene <em>TP53 (p53)</em><em>, </em>but has variable and incomplete penetrance, suggesting additional acquired somatic mutations are necessary for tumorigenesis. In contrast, LFSL has no known cause, although a 10-Mb …

    uthsc Repository record for Omics Approaches to Uncover Germline and Somatic Variation Underlying Inherited Sarcomagenesis (opens in a new tab)

  4. Identifikation von Mutationen im Tumorsuppressorgen p53 und des Bakteriums Helicobacter pylori in Magenkarzinomen histopathologischer Präparate aus verschiedenen medizinhistorischen Sammlungen

    … zu rezenten gibt. Als typisches Zielgen für Tumormutationen wurde das Tumorsuppressorgen p53 untersucht. Au¬ßerdem wurde Helicobacter pylori (H. pylori) als Risikofaktor für die Entstehung von Magenkarzinomen in den historischen Proben identifiziert. Insgesamt standen 51 Gewebeproben von …

    goettingen Repository record for Identifikation von Mutationen im Tumorsuppressorgen p53 und des Bakteriums Helicobacter pylori in Magenkarzinomen histopathologischer Präparate aus verschiedenen medizinhistorischen Sammlungen (opens in a new tab)

  5. Investigation into the molecular mechanisms underlying circadian rhythm disruption and human cancer

    … promoting p53 stability. Critically, one p53 mutation, Y220C (YC), is a hotspot in cancer and uniquely lies at the binding site of PER2 within p53, distant from the interface binding to DNA. We hypothesized that an altered interaction between PER2 and p53YC results in less available PER2 and …

    vt Repository record for Investigation into the molecular mechanisms underlying circadian rhythm disruption and human cancer (opens in a new tab)

  6. Accurate Mutation Annotation and Functional Prediction Enhance The Applicability of -Omics Data In Precision Medicine

    … data sources, and is crucial for precise mutation biomarker identification and functional prediction; 3) It improves “forward annotation” (i.e., translation of genomic variants to protein variants) over existing annotators by more rigorously implementing the Human Genome Variation Society …

    uthsc Repository record for Accurate Mutation Annotation and Functional Prediction Enhance The Applicability of -Omics Data In Precision Medicine (opens in a new tab)

  7. Quantitative dynamics of T-cell receptor repertoires and their use for cancer patient stratification

    … use for patient stratification based on their mutational status for the hotspot mutation IDH1 R132H in glioma. First, in Chapter 2 we deeply characterised the statistical properties of data generated from TCR repertoire sequencing, in order to understand the source and amount of expected …

    cambridge Repository record for Quantitative dynamics of T-cell receptor repertoires and their use for cancer patient stratification (opens in a new tab)

  8. Genetic basis of inherited kidney and related tumours

    … phenotype. This variant occurs as a somatic hotspot mutation in RCC and disrupts pVHL-ELOC interaction mimicking the effects of pVHL inactivation on expression of hypoxia response elements. *ELOC* functions as a tumour suppressor gene and chromosome 8 deletion was demonstrated in the RCC …

    cambridge Repository record for Genetic basis of inherited kidney and related tumours (opens in a new tab)