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Showing 1 to 8 of 8 for “"dipyridamole"”.

  1. Structure-Activity Relationship and Mechanistic Studies on the Chemopreventive Activity of Dipyridamole and Its Analogues

    … and targets. The nucleoside transport inhibitor dipyridamole (<strong>DPM</strong>) showed potent chemopreventive activity against JB6 P<sup>+</sup>cells (tumor promotion sensitive). To probe the effects of <strong>DPM</strong> structural features on its antitumor promotion activity, the …

    tenn-hsc Repository record for Structure-Activity Relationship and Mechanistic Studies on the Chemopreventive Activity of Dipyridamole and Its Analogues (opens in a new tab)

  2. Computer-Aided Drug Design and Discovery, Screening and Synthesis of Small Molecule Inhibitors of Nucleoside Transporters

    … for biological testing. The NBMPR analogue and dipyridamole analogue hENT1 pharmacophores were compared to each other and to a combined pharmacophore for hENT1. The dipyridamole analogue pharmacophore better predicted non-nucleoside small molecule inhibitors, and as such appears to be the better …

    tenn-hsc Repository record for Computer-Aided Drug Design and Discovery, Screening and Synthesis of Small Molecule Inhibitors of Nucleoside Transporters (opens in a new tab)

  3. Investigating Crystallization Tendency, Miscibility and Molecular interactions of drug-polymer systems for the development of amorphous solid dispersions

    … (CUR), indomethacin (IND), flutaminde (FLU), dipyridamole (DIP), griseofulvin (GRI)] in absence and presence of four different polymers [i.e. polyethylene glycol (PEG), eudragit EPO (EPO), hydroxypropyl methylcellulose (HPMC) and polyvinyl pyrrolidone (PVP)] in various drug-polymer ratios were …

    creighton Repository record for Investigating Crystallization Tendency, Miscibility and Molecular interactions of drug-polymer systems for the development of amorphous solid dispersions (opens in a new tab)

  4. Mechanisms of ion channel activation in primary cultured and clonal cell lines

    … B, or generation of membrane tension by dipyridamole failed to elicit significant increases in cell chloride permeability. The mechanism of current activation is as yet undetermined. The currents were effectively inhibited by the chloride channel inhibitors NPPB and DIDS but resistant to …

    aston Repository record for Mechanisms of ion channel activation in primary cultured and clonal cell lines (opens in a new tab)

  5. A study of the binding of anti-aggregatory substances to platelet bound plasma proteins.

    … was placed on the study of the binding of dipyridamole (DPD) and its analogues. The data obtained from the above experiments was compared with that obtained from the classical methods of equilibrium dialysis, dynamic dialysis, and continuous ultrafiltration and used to set up a number of …

    rgu Repository record for A study of the binding of anti-aggregatory substances to platelet bound plasma proteins. (opens in a new tab)

  6. Peri-operative cardiac morbidity: prediction, prevention and the novel role of B-type natriuretic peptide

    … time are dobutamine stress echocardiography and dipyridamole thallium scanning. However they are expensive, time consuming and have shown poor positive predictive ability, even in high risk cohorts. Few studies have studied the usefulness of biochemical markers in the prediction of post-operative …

    glasgow Repository record for Peri-operative cardiac morbidity: prediction, prevention and the novel role of B-type natriuretic peptide (opens in a new tab)

  7. Biochemische Charakterisierung des ADP-Rezeptors P2Y12 und pharmakologische Therapiekontrolle von Thrombozytenfunktionshemmern

    Die Bedeutung der cAMP- und cGMP-abhängigen Proteinkinase für die Hemmung der Plättchenaktivierung und -aggregation ist gut beschrieben. Zahlreiche fundamentale Plättchenantworten wie die Erhöhung der intrazellulären Calciumkonzentration, die Exposition von Adhäsionsrezeptoren und die …

    wurz-thes Repository record for Biochemische Charakterisierung des ADP-Rezeptors P2Y12 und pharmakologische Therapiekontrolle von Thrombozytenfunktionshemmern (opens in a new tab)