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Showing 1 to 8 of 8 for “"cyclooxygenases"”.

  1. Synergistic inhibition of cyclooxygenases and monoacylglycerol lipase in neuropathic pain

    Neuropathic pain is caused by altered nerve function that often presents as allodynia, which is the painful perception of typically non-noxious stimuli. Neuropathic pain is commonly treated with GABA analogues, steroids, or non-steroidal anti-inflammatory drugs (NSAIDs). NSAIDs inhibit one or more …

    wvu Repository record for Synergistic inhibition of cyclooxygenases and monoacylglycerol lipase in neuropathic pain (opens in a new tab)

  2. Expression and Function of a Putative Cox-Like Gene in D. melanogaster

    <p>Cyclooxygenases (COX) are the enzymes that catalyze the conversion of arachidonic acid into prostaglandins. In mammals, isoform COX-1 is constitutively expressed, whereas the isoform COX-2 gene expression is induced, primarily at sites of inflammation. While eicosanoids play a major role in …

    cuny Repository record for Expression and Function of a Putative Cox-Like Gene in D. melanogaster (opens in a new tab)

  3. The mechanism of action of the anticancer effects of selenomethionine on colon cancer

    … might affect cell growth by mechanisms involving cyclooxygenases, specifically the inducible isoform COX-2. Cyclooxygenases (COX-1 and COX-2) are enzymes that metabolize arachidonic acid to various prostaglandins. The human adenocarcinoma cell lines HT-29 and HCA-7, that express variable levels of …

    arizona-thes Repository record for The mechanism of action of the anticancer effects of selenomethionine on colon cancer (opens in a new tab)

  4. Concomitant Suppression Of Both Cox-1 And Cox-2 Is Not Sufficient To Cause Gastroenteropathy Associated With Chronic Nsaid Use

    … consequence of simultaneously inhibiting both cyclooxygenases (COX) -1 and -2 to suppress prostaglandin synthesis. Operating under this assumption, I had developed a novel mouse model of inducible COX-1 + COX-2 deletion to complement our lab’s chronic NSAID exposure model. These COX …

    penn Repository record for Concomitant Suppression Of Both Cox-1 And Cox-2 Is Not Sufficient To Cause Gastroenteropathy Associated With Chronic Nsaid Use (opens in a new tab)

  5. An investigation of the neuroprotective properties of fenamate NSAIDs, against experimental model of ischemic stroke

    … <p>Fenamates are non-selective inhibitors of cyclooxygenases. In addition, fenamates are antagonists of non-selective cation channels, subtype-selective modutators of GABA<sub>A </sub>receptors, weak inhibitors of glutaniate receptors and activators of some potassium channels, all potentially …

    u-pacific Repository record for An investigation of the neuroprotective properties of fenamate NSAIDs, against experimental model of ischemic stroke (opens in a new tab)

  6. Elucidating the Interplay Between Lipids and Membrane Proteins Using Multiscale Computer Simulations

    … interaction profile of AMPA receptors and cyclooxygenases (mainly COX-1), showing that they both form specific interactions with lipids, but do so in a quite different fashion. AMPA receptors interact specifically with diacylglycerol lipids, whereas COX-1 enzymes do so indiscriminately with …

    calgary Repository record for Elucidating the Interplay Between Lipids and Membrane Proteins Using Multiscale Computer Simulations (opens in a new tab)

  7. A Combined Computational Strategy of Sequence and Structural Analysis Predicts the Existence of a Functional Eicosanoid Pathway in <i>Drosophila melanogaster</i>

    … eight low confidence candidates. Four predicted cyclooxygenases and two potential lipoxygenase activating proteins, highly divergent from their human counterparts, were identified, although similar methods failed to identify putative lipoxygenase enzymes. Tertiary structures of a majority of …

    cuny-grad Repository record for A Combined Computational Strategy of Sequence and Structural Analysis Predicts the Existence of a Functional Eicosanoid Pathway in <i>Drosophila melanogaster</i> (opens in a new tab)

  8. Regulation of tumour necrosis factor receptor expression on neutrophils by arachidonic acid and other long chain fatty acids.

    … not sensitive to the action of inhibitors of the cyclooxygenases and lipoxygenases. Using chemical inhibitors of intracellular signaling pathways, we demonstrate that the effect of AA on TNFRI is very sensitive to GFI09203X, PD098059, AACOCF3 and wortmannin, showing a role for protein kinase C, …

    adelaide Repository record for Regulation of tumour necrosis factor receptor expression on neutrophils by arachidonic acid and other long chain fatty acids. (opens in a new tab)