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Showing 1 to 5 of 5 for “"chronic inflammations"”.

  1. Copper and rheumatoid arthritis - an in vitro study of metal-protein interactions

    Acute and Chronic inflammations in Rheumatoid Arthritis are characterized by, among other features, changes in the metabolism of copper. Previous studies have shown that administration of exogenous copper, and the in vivo manipulation of endogenous copper may provide new ways of coping with the …

    cape-town Repository record for Copper and rheumatoid arthritis - an in vitro study of metal-protein interactions (opens in a new tab)

  2. Diffusion and reactions of nitric oxide, oxygen, and superoxide in cells and culture media

    … host tissues, especially if prolonged (as in chronic inflammations). Although NO can act directly, much of its cytotoxicity and genotoxicity may be due to peroxynitrite (ONOO-), formed by the rapid reaction of NO with superoxide (02-)- Superoxide is a byproduct of mitochondrial respiration and …

    mit Repository record for Diffusion and reactions of nitric oxide, oxygen, and superoxide in cells and culture media (opens in a new tab)

  3. Herstellung und Charakterisierung von rekombinanten, ETA-basierten CD64-Immuntoxinen zur Bekämpfung von dysregulierten Makrophagen bei chronischen Hautentzündungen

    … macrophages play a decisive role in different chronically inflammatory cutaneous diseases. The fact that a selective elimination of the high CD64 expressing macrophages, through CD64 immunotoxin (full length antibody with a toxin), results in a resolution of chronic inflammations, confirms …

    aachen Repository record for Herstellung und Charakterisierung von rekombinanten, ETA-basierten CD64-Immuntoxinen zur Bekämpfung von dysregulierten Makrophagen bei chronischen Hautentzündungen (opens in a new tab)

  4. Effects of endotoxin-induced inflammation on glucose metabolism and contractility of the rat heart in vivo and in vitro

    Acute and chronic inflammations of the myocardium (as induced by sepsis) are often associated with a severe impairment of cardiac contractility. Key players in these events are proinflammatory cytokines, such as Tumor Necrosis Factor (TNFa) and Interleukin-1b (IL-1b). Despite the fact (i) that …

    aachen Repository record for Effects of endotoxin-induced inflammation on glucose metabolism and contractility of the rat heart in vivo and in vitro (opens in a new tab)