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Showing 1 to 6 of 6 for “"cFLIP"”.

  1. Characterization of the anti-type I interferon effects of the cellular flice-like inhibitory protein (CFLIP)

    The cellular FLICE-like inhibitory protein (cFLIP) is well known as a major immunomodulatory protein. Various works have described its role in major cellular pathways such as apoptosis, autophagy, necroptosis, NF-KB regulation and more recently, modulation of interferon responses. Interferon alpha …

    uiuc Repository record for Characterization of the anti-type I interferon effects of the cellular flice-like inhibitory protein (CFLIP) (opens in a new tab)

  2. Direct Regulation of Apoptosis By Linear Ubiqutin Chain Assembly Complex (Lubac) and Feedback Regulation of Lubac Function By Caspases

    … cellular FLICE-like inhibitory protein (cFLIP). Finally, I determined the negative feedback to regulate function of LUBAC from apoptosis. Effector caspases induce RNF31 cleavage in apoptotic condition, which suppresses its role in NF-kB activation. This study is the first to demonstrate …

    uthsc Repository record for Direct Regulation of Apoptosis By Linear Ubiqutin Chain Assembly Complex (Lubac) and Feedback Regulation of Lubac Function By Caspases (opens in a new tab)

  3. The Role of Alternative Splicing and Alternative Polyadenylation in Regulating Activated T cell Biology

    … may regulate T cell survival by controlling cFLIP levels through promoting proximal PAS usage within the 3'UTR tail. In summary, this dissertation demonstrates broad alternative splicing and polyadenylation changes are fine-tuned to specific T cell stimuli and exert biological impact on cell …

    penn Repository record for The Role of Alternative Splicing and Alternative Polyadenylation in Regulating Activated T cell Biology (opens in a new tab)

  4. In vivo and in vitro studies of immunodeficiency in Ataxia-telangiectasia

    … for a role of ATM in regulating either CD95 or cFLIP expression. Consistent with this was the increased sensitivity to CD95-mediated apoptosis of T cell prolymphocytic leukaemia (T-PLL) cells. The cause of the tumour is primary loss of ATM activity (either germline loss similar to A-T LCLs or …

    birmingham Repository record for In vivo and in vitro studies of immunodeficiency in Ataxia-telangiectasia (opens in a new tab)

  5. Papel de YAP/TAZ en la respuesta de células tumorales a estímulos activadores de receptores de TRAIL

    … oligomerización TRAIL-R2, y mantiene niveles de cFLIP suficientes para dificultar la activación de la caspasa-8; esto favorece la supervivencia de la célula tumoral a la UPR. Las células tumorales también deben sobrevivir a la escasez de aminoácidos. La glutamina es el primer aminoácido en …

    sevilla Repository record for Papel de YAP/TAZ en la respuesta de células tumorales a estímulos activadores de receptores de TRAIL (opens in a new tab)

  6. Papel del metabolismo de la glutamina en el control de la sensibilidad a trail de células tumorales de mama.

    … demuestran que la reducción de los niveles de cFLIP es el evento responsable de la marcada sensibilización de las células TNBC a la apoptosis inducida por TRAIL tras privación de glutamina. Además, nuestros resultados demuestran que la inhibición farmacológica de las transaminasas aumenta los …

    sevilla Repository record for Papel del metabolismo de la glutamina en el control de la sensibilidad a trail de células tumorales de mama. (opens in a new tab)