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Showing 1 to 20 of 21 for “"Wallerian degeneration"”.

  1. A DLK And JNK Dependent Axon Self-Destruction Program Promotes Wallerian Degeneration

    … sequence of morphological changes termed Wallerian degeneration. It has long been hypothesized that there is an active axonal breakdown program, conceptually similar to apoptosis, which underlies Wallerian degeneration. However, the molecular pathways that accomplish this program in …

    wustl Repository record for A DLK And JNK Dependent Axon Self-Destruction Program Promotes Wallerian Degeneration (opens in a new tab)

  2. Experimental Modelling and Molecular Mechanisms of Wallerian Degeneration in Traumatic Axonal Injury

    … injury mechanism that worsens TAI may be Wallerian degeneration (WD), a cell-autonomous axonal death pathway. The relationship between traumatic axonal injury and WD is poorly characterised. This thesis explores the basic mechanisms by which a physical axonal trauma can lead to WD, and how …

    cambridge Repository record for Experimental Modelling and Molecular Mechanisms of Wallerian Degeneration in Traumatic Axonal Injury (opens in a new tab)

  3. The Apoptotic and Wallerian Degeneration Pathways Regulate Disease Onset and Progression in the SOD-1G93A Transgenic Mouse Model of ALS

    … by progressive muscle denervation, motor axon degeneration and the death of motor neurons. The molecular mechanisms that mediate axon degeneration in ALS remain unknown, but motor neuron cell body death occurs through apoptosis. Genetic deletion of the pro-apoptotic gene Bax delays muscle …

    rockefeller Repository record for The Apoptotic and Wallerian Degeneration Pathways Regulate Disease Onset and Progression in the SOD-1G93A Transgenic Mouse Model of ALS (opens in a new tab)

  4. Vulnerability of ex vivo α-motor nerve terminals to hypoxia-reperfusion injury.

    … but, occurs via a mechanism distinct from Wallerian degeneration, as the neuroprotective slow Wallerian degeneration (Wlds) gene did not protect nerve terminals from these pathological changes. I also provide provisional evidence to show that 1A/II muscle spindle afferents and γ-motor nerve …

    edinburgh Repository record for Vulnerability of ex vivo α-motor nerve terminals to hypoxia-reperfusion injury. (opens in a new tab)

  5. The Heparan Sulfate Proteoglycan Perlecan Regulates Axonal and Synaptic Stability

    … These phenotypes are not prevented by blocking Wallerian degeneration and are independent of Perlecan’s role in Wingless signaling. Overexpression of Perlecan in motoneurons cannot rescue synaptic retraction phenotypes. Similarly, removing Perlecan specifically from neurons, glia, muscle, fat …

    mit Repository record for The Heparan Sulfate Proteoglycan Perlecan Regulates Axonal and Synaptic Stability (opens in a new tab)

  6. THE EFFECTS OF NICOTINAMIDE MONONUCLEOTIDE ADENYLYLTRANSFERASE 2 (NMNAT2) ON MOUSE NERVE AND BLADDER DEVELOPMENT

    … the most widely studied for its ability to delay Wallerian degeneration. Studies have linked the Nmnat1 portion of the Wlds fusion protein to the majority of its ability to protect axons, but recent work has identified Nmnat2 as the endogenous factor involved in axon maintenance. Our goal is to …

    wfu Repository record for THE EFFECTS OF NICOTINAMIDE MONONUCLEOTIDE ADENYLYLTRANSFERASE 2 (NMNAT2) ON MOUSE NERVE AND BLADDER DEVELOPMENT (opens in a new tab)

  7. Diffuse Brain Injury Triggers Ultra-Rapid Perisomatic Traumatic Axonal Injury, Wallerian Change, and Non-Specific Inflammatory Responses

    … ultrastructure revealed the initial stages of Wallerian degeneration. Axotomy sites did not internalize pre-injury administered dextran suggesting pathogenesis independent of altered axolemmal permeability. Given the rapidity of perisomatic axotomy, absence of axolemmal permeability may …

    vcu Repository record for Diffuse Brain Injury Triggers Ultra-Rapid Perisomatic Traumatic Axonal Injury, Wallerian Change, and Non-Specific Inflammatory Responses (opens in a new tab)

  8. A New Type of Programmed Cell Death in C. Elegans

    … for the involvement of autophagic, necrotic, or Wallerian degeneration genes in linker cell death. By ablating cells neighboring the linker cell, and by examining mutants in which the linker cell is abnormally positioned, we demonstrated that the linker cell employs a cell-autonomous program to …

    rockefeller Repository record for A New Type of Programmed Cell Death in C. Elegans (opens in a new tab)

  9. Deposition of Electroactive Polymers onto Polymer Nanofibers for Nerve Regeneration

    … can regenerate after an injury through the Wallerian degeneration process, the degree of regeneration is often not efficient enough to achieve full return of functionality. To overcome the disadvantages of current nerve repair approaches, peripheral nerve tissue engineering strategies have …

    texas-state Repository record for Deposition of Electroactive Polymers onto Polymer Nanofibers for Nerve Regeneration (opens in a new tab)

  10. Exploring SARM1 as a target to delay programmed axon degeneration

    Programmed axon degeneration (Wallerian degeneration) can occur after physical injury, inhibition of axon transport, exposure to neurotoxic compounds, and in diseases involving mitochondrial or metabolic dysfunction. There is increasing evidence that this pathway can be activated in human painful …

    cambridge Repository record for Exploring SARM1 as a target to delay programmed axon degeneration (opens in a new tab)

  11. Axon Degeneration and the Schwann Cell Early Injury Response: A Study in Mouse and Zebrafish

    … axons degenerate through a process termed Wallerian degeneration and Schwann cells transform into a repair phenotype. Axon degeneration is regulated by a signalling pathway controlled by the pro-degenerative axon death molecule sterile-alpha and toll/interleukin 1 receptor motif containing …

    cambridge Repository record for Axon Degeneration and the Schwann Cell Early Injury Response: A Study in Mouse and Zebrafish (opens in a new tab)

  12. Systemic AAV-Mediated Gene Therapy Using Epo-R76E to Protect Retinal Ganglion Cells from Optic Nerve Injury and Disease

    … degenerating neurons in an induced model of neurodegeneration and a spontaneous model of glaucoma.</p> <p>Optic nerve crush is a model of retinal ganglion cell (RGC) death. Crushing the optic nerve causes the axons to undergo Wallerian degeneration and apoptotic loss of retinal ganglion cells over …

    tenn-hsc Repository record for Systemic AAV-Mediated Gene Therapy Using Epo-R76E to Protect Retinal Ganglion Cells from Optic Nerve Injury and Disease (opens in a new tab)

  13. Investigating novel therapeutic approaches and targets to prevent synapse degeneration

    … therapies aimed at preventing or halting neurodegeneration. Despite the continuously growing body of research elucidating the molecular mechanisms that modulate synaptic function and vulnerability, the contribution of these pathways to neurodegenerative diseases is far from fully characterized. …

    edinburgh Repository record for Investigating novel therapeutic approaches and targets to prevent synapse degeneration (opens in a new tab)

  14. Contribution of microglial reactivity to olfactory ensheathing cell migration in vivo

    … experiments determined the time-frame in which Wallerian degeneration (WD) induced microglial reactivity occurs in the right dorsal corticospinal tract (dCST) of adult rats at the level of T11 following aspiration of the contralateral sensorimotor cortex. This timing data from this study …

    sask Repository record for Contribution of microglial reactivity to olfactory ensheathing cell migration in vivo (opens in a new tab)

  15. Mutational and Structural Investigation of SARM1, a Protein that Mediates Axon Loss

    … NADase that executes the programmed axon degeneration pathway after nerve injury and in diseases including polyneuropathies. The ARM domain of SARM1 plays a crucial role in regulating its NADase activity. By attenuating this activity, axon degeneration is delayed, making SARM1 an important …

    cambridge Repository record for Mutational and Structural Investigation of SARM1, a Protein that Mediates Axon Loss (opens in a new tab)

  16. Chronic activation and downstream mechanisms of programmed axon death

    … a conserved, well-characterised pathway of axon degeneration activated by physical injury and in disease states. The two main regulators of the pathway are the pro-survival NAD-synthesising enzyme NMNAT2, and the pro-degenerative NAD(P)-consuming enzyme SARM1. Over-expression of NMNAT enzymatic …

    cambridge Repository record for Chronic activation and downstream mechanisms of programmed axon death (opens in a new tab)

  17. Programmed axon death as a driver of environmental neurotoxicity triggered by pyridine derivatives

    … preventable pathway leading to axon degeneration. Programmed axon death is regulated by SARM1, a pro-degenerative, multi- functional enzyme that consumes NAD and NADP, with dramatic consequences for neuron energy metabolism. Accumulating evidence in both pre-clinical disease models …

    cambridge Repository record for Programmed axon death as a driver of environmental neurotoxicity triggered by pyridine derivatives (opens in a new tab)

  18. Die Rolle des Interleukin-6 bei der Wallerschen Degeneration

    Die Rolle des Interleukin-6 bei der Wallerschen Degeneration. Untersucht wurde mit Hilfe von Interleukin-6 Knockout Mäusen die Rolle des Interleukin-6. Am Modell der Axotomie 4- und 6-Tage nach Axotomie des N. ischiadicus im Vergleich zu Wildtyp-Mäusen. Untersucht wurden immunhistochemisch die …

    goettingen Repository record for Die Rolle des Interleukin-6 bei der Wallerschen Degeneration (opens in a new tab)

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