Global ETD Search

Search theses and dissertations gathered from participating repositories worldwide. Every result links back to the library that holds it. No account is needed.

Results

Showing 1 to 20 of 31 for “"Settore MEDS-02/A - Patologia generale"”.

  1. LINKING TRANSCRIPTIONAL AND MORPHOLOGICAL HETEROGENEITY TO THERAPEUTIC VULNERABILITIES IN GLIOBLASTOMA

    This thesis is the result of my doctoral studies conducted within the Iorio Laboratory at Human Technopole and whose overarching goal has been to design and use data-driven approaches for the integration of molecular and functional-genetics data from preclinical models with the aim of prioritizing …

    milano Repository record for LINKING TRANSCRIPTIONAL AND MORPHOLOGICAL HETEROGENEITY TO THERAPEUTIC VULNERABILITIES IN GLIOBLASTOMA (opens in a new tab)

  2. Targeting LGALS3 to treat steatotic liver disease and hepatocarcinogenesis

    Non-alcoholic fatty liver disease (NAFLD), recently renamed as Metabolic dysfunction-associated steatotic liver disease (MASLD), is the leading cause of chronic liver disease (CLD) and liver-related morbidity and mortality worldwide. NAFLD, characterized by hepatic fat accumulation and coexisting …

    cagliari Repository record for Targeting LGALS3 to treat steatotic liver disease and hepatocarcinogenesis (opens in a new tab)

  3. A NOVEL, RAS-INDEPENDENT ROLE FOR NF1 IN MICROTUBULE DYNAMICS AND DAMAGE REPAIR DICTATES SENSITIVITY TO T-DM1 IN HER2 POSITIVE BREAST CANCER.

    Neurofibromatosis 1 (NF1) is among the first tumor suppressor genes to be cloned. Loss-of-function (LoF) mutations in NF1 are at the basis of the onco-developmental syndrome Neurofibromatosis type 1 and are frequently found at the somatic level across multiple tumors. In breast cancer, NF1 LoF has …

    milano Repository record for A NOVEL, RAS-INDEPENDENT ROLE FOR NF1 IN MICROTUBULE DYNAMICS AND DAMAGE REPAIR DICTATES SENSITIVITY TO T-DM1 IN HER2 POSITIVE BREAST CANCER. (opens in a new tab)

  4. TARGETING TYPE-I PRMTS RESTORES PLATINUM SENSITIVITY IN OVARIAN CARCINOMA THROUGH MITOCHONDRIAL METABOLISM

    … arginine methyltransferases (PRMTs) with MS023 re-sensitizes A2780R cells to carboplatin, reducing IC50 by up to 56%, and abrogates the enhanced mitochondrial activity. MS023 treatment induces depolarization of the mitochondrial membrane, diminishes mitochondrial volume and network …

    milano Repository record for TARGETING TYPE-I PRMTS RESTORES PLATINUM SENSITIVITY IN OVARIAN CARCINOMA THROUGH MITOCHONDRIAL METABOLISM (opens in a new tab)

  5. THE ROLE OF EXTRACELLULAR VESICLES IN HEART DISEASES

    … HFrEF, and HFpEF. HCM diagnosis followed the 2023 ESC criteria, including cardiac imaging, clinical assessment, and genetic screening using next-generation sequencing of 54 sarcomere-related genes. Echocardiographic imaging was performed to assess structural and functional parameters in all …

    milano Repository record for THE ROLE OF EXTRACELLULAR VESICLES IN HEART DISEASES (opens in a new tab)

  6. DISSECTING THE ROLE OF GOLGI TRAFFIC IN BRAIN DEVELOPMENT ACROSS HEALTH AND DISEASE: A LESSON FROM COG5-CDG STUDIES

    Neurons forming the neocortex are generated during embryonic development from two main classes of neural progenitor cells: apical and basal progenitors (APs and BPs, respectively). The cell fate switch from AP to BP is accompanied by changes in polarization and intracellular architecture. …

    milano Repository record for DISSECTING THE ROLE OF GOLGI TRAFFIC IN BRAIN DEVELOPMENT ACROSS HEALTH AND DISEASE: A LESSON FROM COG5-CDG STUDIES (opens in a new tab)

  7. ROLE OF A SPECIFIC NANOG SUPER-ENHANCER ERNA IN EPIGENETIC REMODELING AND ENHANCER-PROMOTER INTERACTION

    During last years, the role of enhancer RNAs has been in part explored; however, it’s still not clear the exact role these transcripts carry out concerning gene regulation. In this work, the role of eRNAs transcribed from the region 5kb upper the TSS of Nanog gene in mESCs has been investigated. In …

    milano Repository record for ROLE OF A SPECIFIC NANOG SUPER-ENHANCER ERNA IN EPIGENETIC REMODELING AND ENHANCER-PROMOTER INTERACTION (opens in a new tab)

  8. EXPLORING THE MUTATIONAL LANDSCAPE OF MITOCHONDRIAL GENOME IN HEMATOLOGIC MALIGNANCIES

    Mitochondria are key regulators of energy metabolism, apoptosis, and redox signaling, and their genome (mtDNA) is particularly vulnerable to genomic alterations due to its proximity to the respiratory chain, lack of protective histones, and limited repair capacity. Although mtDNA mutations have …

    milano Repository record for EXPLORING THE MUTATIONAL LANDSCAPE OF MITOCHONDRIAL GENOME IN HEMATOLOGIC MALIGNANCIES (opens in a new tab)

  9. ENDOCYTIC CONTROL OF CELL-AUTONOMOUS AND NON-CELL-AUTONOMOUS FUNCTIONS OF P53

    NUMB is a multifunctional adaptor protein historically recognized for its role in determining cell fate decisions through inhibition of NOTCH signaling, a process achieved via endocytic trafficking. Subsequently, NUMB has emerged as a significant tumor suppressor, particularly in the context of …

    milano Repository record for ENDOCYTIC CONTROL OF CELL-AUTONOMOUS AND NON-CELL-AUTONOMOUS FUNCTIONS OF P53 (opens in a new tab)

  10. A SEARCH FOR EPIGENETIC VULNERABILITIES OF AML CELLS PERFORMED BY A CDK INHIBITOR¿BASED CRISPR SCREENING

    Acute myeloid leukemia is an aggressive hematologic malignancy characterized by uncontrolled proliferation and impaired differentiation of hematopoietic progenitors. Epigenetic dysregulation plays a central role in AML pathogenesis, and targeting chromatin-modifying enzymes has emerged as a …

    milano Repository record for A SEARCH FOR EPIGENETIC VULNERABILITIES OF AML CELLS PERFORMED BY A CDK INHIBITOR¿BASED CRISPR SCREENING (opens in a new tab)

  11. ASSESSING PREDICTIVE MODELING APPROACHES FOR OPTIMIZING THERAPY IN ADVANCED LARYNGEAL AND HYPOPHARYNGEAL CANCERS

    Background: Laryngeal (LC) and hypopharyngeal (HPC) cancers are often diagnosed at an advanced stage (III or IV) due to the absence of early symptoms and biomarkers. First-line therapies include both surgical and non-surgical approaches. Since surgical removal, comprising total and partial …

    milano Repository record for ASSESSING PREDICTIVE MODELING APPROACHES FOR OPTIMIZING THERAPY IN ADVANCED LARYNGEAL AND HYPOPHARYNGEAL CANCERS (opens in a new tab)

  12. ELUCIDATING THE IMPACT OF ASCITES ON HIGH GRADE SEROUS OVARIAN CANCER THROUGH MULTIPLE IN VITRO PATIENT-SPECIFIC MODELS

    High-grade serous ovarian cancer (HGSOC) is a highly heterogeneous and aggressive malignancy characterized by marked inter-/intra-tumor variability and a dynamic tumor microenvironment (TME). The TME, and particularly ascitic fluid (AF), plays a critical role in OC progression by influencing tumor …

    milano Repository record for ELUCIDATING THE IMPACT OF ASCITES ON HIGH GRADE SEROUS OVARIAN CANCER THROUGH MULTIPLE IN VITRO PATIENT-SPECIFIC MODELS (opens in a new tab)

  13. INSIGHTS INTO THE SELECTION AND FUNCTION OF COPY-NUMBER ALTERATIONS DURING CANCER EVOLUTION

    Cancer evolution is a complex process mostly driven by genomic instability, including large- and small-scale alterations, such as copy-number alterations (CNAs) or single-nucleotide mutations, respectively. Those alterations play a critical role in tumor development, genetic diversity, and …

    milano Repository record for INSIGHTS INTO THE SELECTION AND FUNCTION OF COPY-NUMBER ALTERATIONS DURING CANCER EVOLUTION (opens in a new tab)

  14. DISSECTING THE ROLE OF THE TETRASPANIN MS4A4A IN MODULATING MACROPHAGE PLASTICITY AND FUNCTION

    MS4A4A belongs to the membrane-spanning 4A (MS4A) family, whose proteins show preferential expression in distinct cell types and structural similarities to tetraspanins, including the ability to interact with and modulate the activity of various immunoreceptors, such as pattern recognition …

    milano Repository record for DISSECTING THE ROLE OF THE TETRASPANIN MS4A4A IN MODULATING MACROPHAGE PLASTICITY AND FUNCTION (opens in a new tab)

  15. DISSECTION OF HOST-VIRUS INTERACTIONS IN THE HUMAN CENTRAL NERVOUS SYSTEM

    Viral encephalitis is an inflammatory brain that can lead to severe neurological deficits, despite antiviral treatment. Neurotropic viruses cause viral encephalitis due to their ability to invade the central nervous system (CNS), and the most common cause is HSV-1. Inborn errors of immunity (IEIs) …

    milano Repository record for DISSECTION OF HOST-VIRUS INTERACTIONS IN THE HUMAN CENTRAL NERVOUS SYSTEM (opens in a new tab)

  16. LARGE-SCALE METAGENOMIC ANALYSIS TO IDENTIFY HUMAN GUT MICROBIOME BIOMARKERS OF CARDIOMETABOLIC HEALTH

    The human gut microbiome, a complex community of microorganisms inhabiting the human gastrointestinal tract, is recognized as a critical modulator of host physiology. Imbalances in its composition have been linked with a range of human diseases, including cardiometabolic disorders. Diet is both a …

    milano Repository record for LARGE-SCALE METAGENOMIC ANALYSIS TO IDENTIFY HUMAN GUT MICROBIOME BIOMARKERS OF CARDIOMETABOLIC HEALTH (opens in a new tab)

  17. EVALUATION OF THE RISK OF SECONDARY LEUKEMIAS IN CANCER SURVIVORS

    Therapy-related myeloid neoplasms (t-MNs) are severe late complications of chemotherapy and/or radiotherapy in cancer survivors, characterized by dismal prognosis and resistance to treatment. Chemotherapy may also play a role in increasing clonal hematopoiesis (CH), a clinical entity associated …

    milano Repository record for EVALUATION OF THE RISK OF SECONDARY LEUKEMIAS IN CANCER SURVIVORS (opens in a new tab)

  18. EXPLORING THE PRE-LEUKEMIC PHASE OF ACUTE PROMYELOCYTIC LEUKEMIA: INSIGHTS FROM STUDIES OF SINGLE CELL ¿OMICS¿

    Acute Promyelocytic Leukemia (APL) is a distinct subtype of Acute Myeloid Leukemia (AML) driven by the oncogenic fusion protein PML-RAR, which acts as an aberrant transcription factor and blocks myeloid maturation at the promyelocyte stage. In vivo, the expression of PML-RAR is necessary but not …

    milano Repository record for EXPLORING THE PRE-LEUKEMIC PHASE OF ACUTE PROMYELOCYTIC LEUKEMIA: INSIGHTS FROM STUDIES OF SINGLE CELL ¿OMICS¿ (opens in a new tab)

  19. MULTI-ORGAN FAILURE DRIVEN BY TELOMERE UNCAPPING

    The telomere-bound shelterin complex safeguards chromosome ends from being recognized as DNA double-strand breaks, a function mostly dependent on TRF2. Loss of TRF2 results in telomere uncapping, activation of the DNA damage response, and chromosomal end-to-end fusions, with dramatic consequences …

    milano Repository record for MULTI-ORGAN FAILURE DRIVEN BY TELOMERE UNCAPPING (opens in a new tab)

Page 1 of 2