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Showing 1 to 4 of 4 for “"Salmonella, SPI1"”.
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The Role of Periplasmic Disulfide Bond Status in the Regulation of the Salmonella SPI1 Type Three Secretion System
Salmonella enterica serovar Typhimurium injects a set of effector proteins into the host cell cytoplasm via the Salmonella Pathogenicity Island I (SPI1) type III secretion system (T3SS) to induce inflammatory diarrhea and bacterial uptake into intestinal epithelial cells. The master SPI1 regulatory …
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Integration of small RNAs in the control of Salmonella pathogenicity island 1(SPI1)
Salmonella induces inflammatory diarrhea and epithelial invasion using a Type Three Secretion System (T3SS) encoded on Salmonella pathogenicity island 1 (SPI1). The SPI1 T3SS directly injects several effector proteins into host cytosols, resulting in host actin rearrangement and engulfment of the …
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Mechanisms of signal integration: Regulation of the SPI1 T3SS
Salmonella must rapidly adapt to various niches in the host during infection. Relevant virulence factors must be appropriately induced, and systems that are detrimental in a particular environment must be turned off. Salmonella infects intestinal epithelial cells using a type 3 secretion system …
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A tale of two proteins: relationship between HilD & FliZ in SPI1 regulation
Submission published under a 24 month embargo labeled 'Closed Access', the embargo will last until 2027-08-01