Global ETD Search

Search theses and dissertations gathered from participating repositories worldwide. Every result links back to the library that holds it. No account is needed.

Results

Showing 1 to 8 of 8 for “"Qki"”.

  1. Qki-Mediated Cholesterol Biosynthesis In Eye Lens and Myelin of The Central Nervous System

    … of cholesterol biosynthesis. We found Quaking (Qki) is a novel transcriptional activator of cholesterol biosynthesis. Using transcriptomic profiling, we found cholesterol biosynthesis was the most downregulated pathway in Qki-depleted lens and brain. Indeed, mRNA and protein levels of …

    uthsc Repository record for Qki-Mediated Cholesterol Biosynthesis In Eye Lens and Myelin of The Central Nervous System (opens in a new tab)

  2. Qki-Mediated Cholesterol Biosynthesis In Eye Lens and Myelin of The Central Nervous System

    … of cholesterol biosynthesis. We found Quaking (Qki) is a novel transcriptional activator of cholesterol biosynthesis. Using transcriptomic profiling, we found cholesterol biosynthesis was the most downregulated pathway in Qki-depleted lens and brain. Indeed, mRNA and protein levels of …

    uthsc Repository record for Qki-Mediated Cholesterol Biosynthesis In Eye Lens and Myelin of The Central Nervous System (opens in a new tab)

  3. Investigating The Role of Quaking In Antigen Uptake and Cross-Presentation By Dendritic Cells

    … discovered that an RNA-binding protein, Quaking (QKI) plays a pivotal role in antigen uptake by DCs. Our previous studies in neural stem cells and microglia have identified QKI as a novel regulator of phagosomes and endolysosomes, and knocking down of QKI significantly downregulates genes involved …

    uthsc Repository record for Investigating The Role of Quaking In Antigen Uptake and Cross-Presentation By Dendritic Cells (opens in a new tab)

  4. Investigating Potential Multi-Protein Complexes Involving Emx2

    … two Emx2-interacting proteins, Cnot6l and QkI (which exists in three forms) that interact with each other. These interactions were initially identified by a yeast two-hybrid screen and then confirmed by pull-down assays. The goal of this project is to use GST pull-down assays determine …

    central-wash Repository record for Investigating Potential Multi-Protein Complexes Involving Emx2 (opens in a new tab)

  5. Confirmation of Emx2 Protein Binding Partners

    … protein-protein interactions between Emx2 and QkI-7, Emx2 and Cnot6l, and Cnot6l and QkI-7. The results were positive for all three interactions. Additionally, Emx2 was found to bind the two other QkI isoforms, QkI-5 and QkI-6.

    central-wash Repository record for Confirmation of Emx2 Protein Binding Partners (opens in a new tab)

  6. Co-regulation of the Smooth Muscle Cell Alternative Splicing Programme by the RNA-Binding Proteins RBPMS, RBFOX2, MBNL1, and QKI

    … In this study, I investigated RBFOX2, MBNL1, and QKI as potential co-regulators of RBPMS in VSMC AS regulation. RBFOX2 was identified as a key candidate due to enrichment of its GCAUG binding motifs around RBPMS-regulated exons in positions suggesting cooperative regulation. MBNL1 was selected for …

    cambridge Repository record for Co-regulation of the Smooth Muscle Cell Alternative Splicing Programme by the RNA-Binding Proteins RBPMS, RBFOX2, MBNL1, and QKI (opens in a new tab)

  7. NF-YA ISOFORMS ROLES IN CLAUDIN LOW CANCERS

    … NF-YA alternative splicing, among which RBFOX2, QKI and CELF2 both in BRCA and in STAD cell lines. Through transient overexpression, I validated RBFOX2 as splicing factor promoting the maturation of NF-YAl. Finally, through a frameshift mutation in NF-YA gene, I have investigated the effect of …

    milano Repository record for NF-YA ISOFORMS ROLES IN CLAUDIN LOW CANCERS (opens in a new tab)

  8. Dissecting the genomic and epigenomic alterations underlying glioblastoma

    … target genes regulating NSC identity, including QKI, PTPRZ1 and CDK6. Intriguingly, SOX2/SOX9 co-binding also target their own and each other’s regulatory regions, suggesting the existence of an auto- and cross-regulatory feed-forward axis. Importantly, we observed no increased in mutational …

    edinburgh Repository record for Dissecting the genomic and epigenomic alterations underlying glioblastoma (opens in a new tab)