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Showing 1 to 6 of 6 for “"Pi3kinase"”.

  1. U0126, MEK1/2 inhibitor, Induces Adhesion Molecules Expression in Human Blood Cells

    박, 성례2011

    … inhibitors such as LY294002 (an inhibitor for PI3Kinase), trolox (antioxidant), rottlerin (an inhibitor for PKC-δ), and SN50 (an inhibitor for NF-kB) on U0126-induced adhesion molecule expression. Adhesion molecule expression was attenuated by rottlerin and trolox. In addition, to examine cell …

    ajou Repository record

  2. Funktionelle Charakterisierung von Daughter of Sevenless

    Bausenwein, BurkhardUniversität Würzburg

    … Untereinheit der Phosphatidylinositol-3-Kinase (PI3Kinase) und der Corkscrew (Csw) Tyrosin Phosphatase wurden durch den Austausch des für die Bindung wichtigen Tyrosins gegen ein Phenylalanin mutiert. Die ektopische Expression der mutierten Konstrukte ohne Bindungsstellen für die Shc, PLCg und …

    wurz-thes Repository record

  3. Regulation of TNF-alpha gene expression by TNFAIP1 as an activator or suppressor in response to lipopolysaccharide

    Aljahdali, Bushra Hameed2019

    … production in early stages is: LPS/ TNFAIP1/ PI3Kinase/AP-1/ TNF-alpha. 6. TNFAIP1 acts as a suppressor in later stage to down-regulate TNF-α gene expression via the following signaling pathway: LPS/TNF-alpha/ TNFAIP1/ Caspase 1/ Apoptotic genes/ Degradation of TNF-α/ Cell apoptosis.

    bu Repository record

  4. Gammabestrahlung führt zur CD95-abhängiger Invasivität in apoptoseresistenten Glioblastomzellen

    Kleber, SusanneUniversität Heidelberg

    … Migration läuft über die Aktivierung der PI3Kinase (PI3K), der Integrin-Linked-Kinase (ILK), der Inhibierung von GSK3 und der Expression der Matrix-Metalloproteinasen (MMPs). Caspasen sind an diesem Signalweg nicht beteiligt. Das direkte Verbindungsglied unterhalb des CD95-Rezeptors …

    heid-diss Repository record

  5. Rolle des PI3K-Signalweges bei der Regulation von Todesrezeptor-induzierter Apoptose in Neuroblastomzellen

    Schneider, MaxiUniversität Ulm

    ulm-diss Repository record

  6. Molecular mechanisms of TOM1-mediated protein trafficking in health and disease

    Roach, Tiffany GraceVirginia Tech2024

    … is the EGFR able to continue signaling, but the PI3Kinase/AKT pathway also gets activated, forcing the cell to survive despite infection. The second mechanism explored originates from a naturally occurring mutation in the TOM1 gene that produces an aspartic acid in position 307 instead of the …

    vt Repository record