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Showing 1 to 5 of 5 for “"PERK signalling"”.

  1. Biochemical and Biophysical Studies of the UPR Kinase PERK and its Interactors in Health and Disease

    … of endoplasmic reticulum (ER) stress. The PERK-eIF2α branch of the UPR is activated by the accumulation of misfolded proteins in the ER lumen. PERK, upon activation through autophosphorylation, recruits its substrate eIF2α for phosphorylation, initiating a transcriptional and translational …

    cambridge Repository record for Biochemical and Biophysical Studies of the UPR Kinase PERK and its Interactors in Health and Disease (opens in a new tab)

  2. Biochemical Studies of the ER Resident Kinase PERK and Implications for Local Translational Control

    … R (PKR)-like endoplasmic reticulum kinase (PERK) is able to couple ER stress to translational repression and transcriptional reprogramming through phosphorylation of eIF2α. This is achieved through the recruitment of eIF2α by a unique cytoplasmic loop in PERK’s kinase domain. PERK signalling

    cambridge Repository record for Biochemical Studies of the ER Resident Kinase PERK and Implications for Local Translational Control (opens in a new tab)

  3. Nutraceutical modulation of lipotoxicity and the development of a tool to monitor palmitate induced sub-cellular dysfunction.

    … long chain ceramide concentrations, through PERK signalling. This provides a possible combining mechanism for curcumins anti-inflammatory and cytotoxicity phenotypes. Lastly (Chapter 6), a protocol for monitoring the sub-cellular relocalisation of proteins and lipids is developed using the …

    cambridge Repository record for Nutraceutical modulation of lipotoxicity and the development of a tool to monitor palmitate induced sub-cellular dysfunction. (opens in a new tab)

  4. Oncogenic KRAS drives heterogeneity in metabolic and signalling pathways through genetic and non-genetic mechanisms

    … is often an activating mutation in key cell signalling hub KRAS. Despite extensive research into PDAC and KRAS, the impact of both genetic and non-genetic variability during the earliest steps of carcinogenesis are scantly described. We hypothesise that both forms of heterogeneity might alter …

    cambridge Repository record for Oncogenic KRAS drives heterogeneity in metabolic and signalling pathways through genetic and non-genetic mechanisms (opens in a new tab)

  5. Investigation of T cell signalling events regulating immunity and tolerance in vivo

    … and quantitative differences in T cell signalling may underlie the differential functional outcomes of priming and tolerance. However, the majority of these studies have relied upon biochemical assessment of signalling in T cell lines or clones, at the population level following …

    glasgow Repository record for Investigation of T cell signalling events regulating immunity and tolerance in vivo (opens in a new tab)