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Showing 1 to 20 of 30 for “"Oncogene Proteins"”.

  1. Small Molecule Regulator of ENTPD5, and ER Enzyme in the PTEN/AKT Pathway

    PI3K signaling plays a crucial role in effecting alterations in a broad range of cellular functions in response to diverse extracellular stimuli (insulin, growth factors, integrins and GPCRs etc.). A key downstream effector of PI3K is the serine-threonine kinase Akt, which in response to PI3K …

    utswmed Repository record for Small Molecule Regulator of ENTPD5, and ER Enzyme in the PTEN/AKT Pathway (opens in a new tab)

  2. The Endoplasmic Reticulum Udpase ENTPD5 Promotes Cancer Cell Growth and Survival in the PI3K/PTEN

    … of protein folding and accumulation of unfolded proteins in the ER and finally lead to ER stress. How does cancer cell solve this problem of increased folding during rapid growth to avoid ER stress? We discovered that ENTPD5, an endoplasmic reticulum (ER) enzyme, is up-regulated in cell lines and …

    utswmed Repository record for The Endoplasmic Reticulum Udpase ENTPD5 Promotes Cancer Cell Growth and Survival in the PI3K/PTEN (opens in a new tab)

  3. Evaluation of Chronic RalGTPase Activation as a Core Specifier of Oncogenic Transformation

    … tumorigenicity of cancers that are driven by oncogenes other than Ras. In order to begin to understand how the Ral pathway may be chronically engaged in diverse oncogenic backgrounds, we further examined the expression of RalGEFs in a variety of cells derived from different tissue origin. Our …

    utswmed Repository record for Evaluation of Chronic RalGTPase Activation as a Core Specifier of Oncogenic Transformation (opens in a new tab)

  4. The Role of Protocadherin 7 in Lung Adenocarcinoma

    Lung cancer is the leading cause of cancer-associated deaths worldwide. Despite recent advances in the development of targeted therapies for lung cancer, most patients develop resistance to these targeted therapies. Our lab has uncovered a key oncogenic role for Protocadherin 7 (PCDH7) in non-small …

    tdl Repository record for The Role of Protocadherin 7 in Lung Adenocarcinoma (opens in a new tab)

  5. The Role of Protocadherin 7 in Lung Adenocarcinoma

    Lung cancer is the leading cause of cancer-associated deaths worldwide. Despite recent advances in the development of targeted therapies for lung cancer, most patients develop resistance to these targeted therapies. Our lab has uncovered a key oncogenic role for Protocadherin 7 (PCDH7) in non-small …

    utswmed Repository record for The Role of Protocadherin 7 in Lung Adenocarcinoma (opens in a new tab)

  6. Novel Functions of the Transcription Factor Aryl Hydrocarbon Receptor (AHR) and Its Tryptophan-Derived Ligands in Cancer Cells

    … of cancer cells, focusing on the proto-oncogene MYC and the aryl hydrocarbon receptor (AHR). The role of MYC in regulating tryptophan (Trp) metabolism was investigated in colon and liver cancer cells using high-performance liquid chromatography-tandem mass spectrometry (LC-MS/MS). Our …

    utswmed Repository record for Novel Functions of the Transcription Factor Aryl Hydrocarbon Receptor (AHR) and Its Tryptophan-Derived Ligands in Cancer Cells (opens in a new tab)

  7. Molecular Mechanisms Underlying Innate Immune Kinase TBK1-Driven Oncogenic Transformation

    … immune response, re-exposure to glucose, or oncogene activation, TBK1 is recruited to the exocyst, where it activates AKT. In cells lacking TBK1, insulin activates AKT normally, but AKT activation by these exocyst-dependent mechanisms is impaired. Discovery and characterization of a …

    utswmed Repository record for Molecular Mechanisms Underlying Innate Immune Kinase TBK1-Driven Oncogenic Transformation (opens in a new tab)

  8. Molecular Regulation of Direct Cardiac Reprogramming

    A heart attack (also known as myocardial infarction, MI) happens when the flow of blood to the heart is blocked. A massive heart attack can kill billions of cardiomyocytes. The heart has limited regenerative potential because adult mammalian cardiomyocytes cannot proliferate, therefore lost …

    utswmed Repository record for Molecular Regulation of Direct Cardiac Reprogramming (opens in a new tab)

  9. Behavioral, Neurochemical, and Histological Characterization of Mice Deficient for Parkin, DJ-1, and Antioxidant Proteins

    … We predicted that reducing levels of antioxidant proteins in parkin-/-DJ-1-/- mice would result in age-dependent nigral cell loss, striatal dopamine depletion or behavioral abnormalities. Characterization of these mice for behavioral abnormalities, neurotransmitter defects and neuropathology, …

    utswmed Repository record for Behavioral, Neurochemical, and Histological Characterization of Mice Deficient for Parkin, DJ-1, and Antioxidant Proteins (opens in a new tab)

  10. Validation of AXL as a Therapeutic Target in Pancreatic Cancer

    Pancreatic ductal adenocarcinoma (PDA), a leading cause of cancer-related death in the US, has a high metastatic rate that is associated with persistent immune suppression. AXL, a member of the TAM (TYRO3, AXL, MERTK) receptor tyrosine kinase family, has been identified as a critical factor that …

    utswmed Repository record for Validation of AXL as a Therapeutic Target in Pancreatic Cancer (opens in a new tab)

  11. Investigating the Functional Contributions and Tumorigenic Potential of Cancer/Testis Antigens and Their Interacting Partners

    The general metadata -- e.g., title, author, abstract, subject headings, etc. -- is publicly available, but access to the submitted files is restricted to UT Southwestern campus access and/or authorized UT Southwestern users.

    utswmed Repository record for Investigating the Functional Contributions and Tumorigenic Potential of Cancer/Testis Antigens and Their Interacting Partners (opens in a new tab)

  12. Molecular Players in Lupus: Leads from Proteomic Screens

    Systemic Lupus Erythematosus is a multifactorial systemic autoimmune disorder marked by anti-nuclear antibodies (ANA), rashes and photosensitivity, joint inflammation, nephritis, and other clinical criteria. SLE develops through the breakdown of three major checkpoints: adaptive immune tolerance, …

    utswmed Repository record for Molecular Players in Lupus: Leads from Proteomic Screens (opens in a new tab)

  13. Epithelial Tubular Fusion, Measuring Interconnection, and the Quest for Solving the Continuity Problem

    The formation of functional epithelial tubules is a central feature of many organ systems. Although it has been well-studied how epithelial cells form tubules, it is not well understood how tubules and related epithelial micro-tissues such as spheroids and organoids interconnect with each other to …

    utswmed Repository record for Epithelial Tubular Fusion, Measuring Interconnection, and the Quest for Solving the Continuity Problem (opens in a new tab)

  14. Ubiquitination of EpoR and p85 in Ligand Induced EpoR Down-Regulation

    … cytoplasmic tyrosines of the EpoR. Signaling proteins bind these phosphotyrosines through SH2 domains, leading to the survival and proliferation of erythroid progenitor cells and the differentiation of these progenitors into mature erythrocytes. Therefore, EpoR signaling is essential for red …

    utswmed Repository record for Ubiquitination of EpoR and p85 in Ligand Induced EpoR Down-Regulation (opens in a new tab)

  15. Identification and Characterization of a Selective Inhibitor of the PIM-1 Kinase

    <p>The PIM-1 kinase is a serine/threonine kinase that has been implicated in the development of many human cancers, including leukemias, lymphomas, and prostate cancer. We have endeavored to identify and characterize a selective inhibitor of the PIM- 1 kinase. Such an inhibitor would have utility …

    loma-linda Repository record for Identification and Characterization of a Selective Inhibitor of the PIM-1 Kinase (opens in a new tab)

  16. Investigating the Role of Complex I Loss in Thyroid Tumorigenesis

    Changes in cellular metabolism are considered one of the hallmarks of cancer. In 1924, Otto Warburg initially posited that defects in cellular respiration drive tumorigenesis after observing that tumor cells often perform aerobic glycolysis in culture. Since then, multiple studies have demonstrated …

    utswmed Repository record for Investigating the Role of Complex I Loss in Thyroid Tumorigenesis (opens in a new tab)

  17. Oncogene-Induced Signaling Heterogeneity in Lung Cancer

    … has identified 138 frequently occurring driver oncogenes and tumor suppressor genes in lung cancer. Currently, only 15 of these genes can be targeted therapeutically. Study of downstream signaling alterations of these oncogenes and tumor suppressor genes may identify novel therapeutic targets. …

    utswmed Repository record for Oncogene-Induced Signaling Heterogeneity in Lung Cancer (opens in a new tab)

  18. Identification and Characterization of Novel Mechanisms of Functional and Structural Synapse Remodeling: Focus on Vav Guanine Nucleotide Exchange Factors and MEF2 Transcription Factors

    … elimination, suggesting that these autism-linked proteins coordinate transcriptional and translational control of common transcripts to mediate proper synaptic connectivity. Using high throughput sequencing of RNA isolated by cross-linking immunoprecipitation (HITS-CLIP) of FMRP, I find a large …

    utswmed Repository record for Identification and Characterization of Novel Mechanisms of Functional and Structural Synapse Remodeling: Focus on Vav Guanine Nucleotide Exchange Factors and MEF2 Transcription Factors (opens in a new tab)

  19. Identification of Oncogenic KRAS-Associated Vulnerabilities in Non-Small Cell Lung Cancer

    Activating mutations in KRAS are frequently involved in the pathogenesis of non-small cell lung cancer (NSCLC), the disease responsible for the most cancer-related deaths in the US. Despite intensive efforts to develop drugs that directly interfere with KRAS activity over the past decade, no …

    utswmed Repository record for Identification of Oncogenic KRAS-Associated Vulnerabilities in Non-Small Cell Lung Cancer (opens in a new tab)

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