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Showing 1 to 4 of 4 for “"Neutrophil survival"”.

  1. The effect of human cytomegalovirus on neutrophil survival, autophagy, and extracellular traps

    Neutrophils provide a rapid first response to invading pathogens and orchestrate the immune response. They are able to employ potent antipathogenic mechanisms such as phagocytosis, reactive oxygen species (ROS) generation, protease release from granules, and formation of neutrophil extracellular …

    cambridge Repository record for The effect of human cytomegalovirus on neutrophil survival, autophagy, and extracellular traps (opens in a new tab)

  2. Human Cytomegalovirus and the Neutrophil

    … and release of new virions, but not the neutrophil. This cell is the first immune cell to engage most pathogens, engulfing and killing them before undergoing apoptosis and clearance by macrophages. However certain viruses and bacteria are able to evade host defences and use the neutrophil

    cambridge Repository record for Human Cytomegalovirus and the Neutrophil (opens in a new tab)

  3. Roles of Neutrophil Activation in Asthma

    Background: Neutrophilic inflammation has been implicated in allergic asthma as well as occupational asthma, which is usually associated with severe phenotype. Autophagy plays crucial roles in innate as well as adaptive immune responses by contributing to the proliferation, activation and …

    ajou Repository record for Roles of Neutrophil Activation in Asthma (opens in a new tab)

  4. Identification of the role of the sal locus in streptococcus pyogenes virulence during host-pathogen interactions

    … to intracellular and extracellular killing by neutrophils. This was most likely a consequence of the high susceptibility of the ΔsalKR mutant to cell lysis by lysozyme, attributable to the differences observed in cell wall modification between the wild type and the mutant. Our data suggests …

    wayne-thes Repository record for Identification of the role of the sal locus in streptococcus pyogenes virulence during host-pathogen interactions (opens in a new tab)