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Showing 1 to 13 of 13 for “"Medical Genetics and Genomics (including Gene Therapy)"”.

  1. Molecular and functional studies of ABL1 and FGFR1 fusion oncogenes in myeloproliferative neoplasms

    The tyrosine kinase encoding genes ABL1 and FGFR1 are involved in fusion genes underlying the myeloproliferative neoplasms chronic myeloid leukemia (CML) and the 8p11-myeloproliferative syndrome (EMS). CML and EMS are both myeloproliferative disorders with an initiating, relatively indolent, …

    lund Repository record for Molecular and functional studies of ABL1 and FGFR1 fusion oncogenes in myeloproliferative neoplasms (opens in a new tab)

  2. EWSR1 and FUS fusion genes in tumorigenesis

    … present thesis concerns the involvement of EWSR1 and FUS fusion genes, and the chimeric proteins they encode, in tumorigenesis. In Article I, the EWSR1 promoter, which regulates the expression of EWSR1 fusion genes, was characterized and regions that were crucial for promoter activity could be …

    lund Repository record for EWSR1 and FUS fusion genes in tumorigenesis (opens in a new tab)

  3. Genetic Characterization of Pediatric T-cell Acute Lymphoblastic Leukemia

    The aim of my thesis has been to characterize genetically pediatric T-cell acute lymphoblastic leukemia (T-ALL). Articles I and II focus on molecular characterization of translocations involving T-cell receptor (TCR) loci. These types of aberration are characteristic for T-ALL and have previously …

    lund Repository record for Genetic Characterization of Pediatric T-cell Acute Lymphoblastic Leukemia (opens in a new tab)

  4. Chromosome dynamics and genomic instablity in neuroblastoma. Three genomic pillars: MYCN amplification, numerical and structural changes.

    … cancer neuroblastoma, one of the most common and lethal childhood tumours. Neuroblastoma has througout the years continued to be a clinical and biological enigma. Our first focus was on one of the most important biological risk factors in neuroblastoma -- amplification of the oncogene MYCN in …

    lund Repository record for Chromosome dynamics and genomic instablity in neuroblastoma. Three genomic pillars: MYCN amplification, numerical and structural changes. (opens in a new tab)

  5. Identification, Validation and Implementation of Blastemal Biomarkers in Wilms Tumour

    … cancer. Blastema is, together with epithelium and stroma, one of the three common histological elements of WT. WTs dominated by blastema after preoperative chemotherapy are classified as high risk tumours. According to the SIOP2001 protocol used in most European countries today, there is no …

    lund Repository record for Identification, Validation and Implementation of Blastemal Biomarkers in Wilms Tumour (opens in a new tab)

  6. Functional Modeling of Genes Upregulated in Chronic Myeloid Leukemia

    … hematopoietic cell by the BCR/ABL1 fusion gene that is formed through the chromosomal translocation t(9;22). CML is currently successfully treated with tyrosine kinase inhibitors targeting the ABL1 kinase domain. However, the CML stem cells are insensitive to this drug and a large fraction …

    lund Repository record for Functional Modeling of Genes Upregulated in Chronic Myeloid Leukemia (opens in a new tab)

  7. IKAROS and LEUKEMIA

    … of immature lymphoid cells in the bone marrow and is the most common cancer type in children. It is an immunophenotypically, morphologically, clinically, and genetically heterogeneous disorder that comprises several distinct subtypes. Proper classification is important because determining the …

    lund Repository record for IKAROS and LEUKEMIA (opens in a new tab)

  8. Trisomies in Hematologic Malignancies

    … aberrations, closely associated with leukemogenesis, are found in many hematologic malignancies. Although the balanced rearrangements, such as translocations and inversions, are the ones most commonly thought of in the context of leukemias, gains of chromosomes ? e.g.., trisomies ? are also …

    lund Repository record for Trisomies in Hematologic Malignancies (opens in a new tab)

  9. Genomic characterization of ETV6/RUNX1-positive acute lymphoblastic leukemia

    The t(12;21) translocation generates the ETV6/RUNX1 fusion gene, present in 25% of childhood acute lymphoblastic leukemia. This fusion gene is important for leukemia development but is not sufficient for leukemia to arise. Hence, the additional genetic changes present in leukemic …

    lund Repository record for Genomic characterization of ETV6/RUNX1-positive acute lymphoblastic leukemia (opens in a new tab)

  10. Mitochondrial and chromosomal genomics in type 2 diabetes

    Understanding the mechanisms of complex polygenic diseases like type 2 diabetes (T2D) is as complex as their nature. Cellular processes involved in glucose homeostasis need high and reliable energy supply. Mitochondria are the major energy producers in mammalian cells. Since majority of the …

    lund Repository record for Mitochondrial and chromosomal genomics in type 2 diabetes (opens in a new tab)

  11. Genetic and epigenetic characterization of pediatric high hyperdiploid acute lymphoblastic leukemia

    The aim of this thesis was to analyze the genetic and epigenetic characteristics of pediatric high hyperdiploid acute lymphoblastic leukemia (HeH ALL), the most common type of childhood malignancy. The three original articles presented in this thesis have addressed three major questions regarding …

    lund Repository record for Genetic and epigenetic characterization of pediatric high hyperdiploid acute lymphoblastic leukemia (opens in a new tab)

  12. Mechanisms and Consequences of Chromosomal Instability in Malignant tumours

    … in established colorectal cancer cell lines and in Wilms tumour. In colorectal cancer cell lines, anaphase bridging was observed to generate both numerical and structural chromosomal aberrations and was also associated with the presence of multipolar mitoses. In contrast to cells having …

    lund Repository record for Mechanisms and Consequences of Chromosomal Instability in Malignant tumours (opens in a new tab)

  13. MOLECULAR PROFILING OF UROTHELIAL CARCINOMA

    The general aim of this thesis was to molecularly characterize urothelial carcinoma (UC) at the transcriptional level using gene expression microarrays to improve the classification and pathogenetic understanding of this disease. In the first two studies (Articles I and II), gene expression …

    lund Repository record for MOLECULAR PROFILING OF UROTHELIAL CARCINOMA (opens in a new tab)