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Showing 1 to 6 of 6 for “"Interleukin-1ß"”.

  1. Acute And Persistent Effects Of Interleukin-1ß And Stress On The HPA Axis

    Tumminello, Tara2014

    <p>.</p>

    shu-thes Repository record

  2. Modulation der Expression von Integrin-Beta4, Interleukin-1Beta und HMGA in humanen kolorektalen Karzinomzellen durch Butyrat

    Burzer, KlausUniversität Würzburg

    … der Proteine Integrin-ß4 (Zelladhäsionsfaktor), Interleukin-1ß (Entzündungsfaktor) und HMGA (Transkriptionsfaktor) ab 48 Stunden Inkubationszeit mit 2mmol/l Butyrat in der gut-differenzierten HT-29-, der entdifferenzierten SW-480-Kolonadenokarzinomzelllinie sowie deren Metastasenzelllinie SW-620 …

    wurz-thes Repository record

  3. Borrelienneuropathie - eine histologische und immunhistochemische Charakterisierung

    Neukirchen, SebastianUniversität Würzburg

    … Zytokine Tumor Nekrose-Faktor-alpha (TNF-alpha), Interleukin-1ß (IL-1ß) und Interleukin-6 (IL-6), als auch Metalloproteinase MMP-9 durchgeführt, ferner mit Antikörpern gegen das membranassoziierte Glykoprotein HLA-DR3. Zusätzlich wurden Semi-Dünn-Schnitte angefertigt. Klinisch wiesen die meisten …

    wurz-thes Repository record

  4. Periventricular white matter damage in the post-natal brain in hypoxic conditions

    DENG YIYU2009

    … upregulated tumor necrosis factor-a (TNF-a), interleukin-1ß (IL-1ß), monocyte chemoattractant protein-1 (MCP-1), chemokine (C-C motif) receptor 2 (CCR2), and macrophage-colony stimulating factor (M-CSF) expression was observed. Immunoexpression of these was specifically located in AMC from …

    nus Repository record

  5. Molecular Dynamics Simulations on the Interleukin-1β:Interleukin Receptor I:Interleukin-1 Receptor Accessory Protein Ternary Complex

    Joshi, Reva

    gmu

  6. MAPK and pro-inflammatory mediators in the walls of brain blood vessels following cerebral ischemia

    Maddahi, AidaLund University, Faculty of Medicine2012

    … expression of some of the major cytokines (IL-1ß, IL-6, TNF-α, TNF-R1 and R2), of MMP-9 (BBB associated) and of iNOS (potential toxic molecule) in cerebral vessel walls. Increased levels and activation of these factors may lead to exacerbation of vasoconstriction, resulting in decreased CBF and …

    lund Repository record