Global ETD Search

Search theses and dissertations gathered from participating repositories worldwide. Every result links back to the library that holds it. No account is needed.

Results

Showing 1 to 4 of 4 for “"HSVtk"”.

  1. Development of a Lentivirus-mediated Gene Therapy Targeting HIV-1 RNA to Eliminate Infected Cells

    … functionalize a defective cell suicide enzyme (*HSVtk*<sub>ΔAUG1</sub>) through targeted RNA *trans*-splicing with the HIV-1 D4 splice site. In-frame *HSVtk* translation is initiated from the start codon of the HIV-1 *tat1* donor exon in chimeric mRNA. HSVtk activates the prodrug ganciclovir …

    cambridge Repository record for Development of a Lentivirus-mediated Gene Therapy Targeting HIV-1 RNA to Eliminate Infected Cells (opens in a new tab)

  2. Mechanisms of Cell Death Initiated in Herpes Simplex Virus Thymidine Kinase Expressing Colon Tumor Cells Treated with Ganciclovir and UCN-01

    … of Herpes Simplex Virus Thymidine Kinase (HSVtk) in combination with the prodrug, ganciclovir (GCV), has shown great promise for the genetic therapy of many cancers, but most patients have had only a partial or minimal response to the therapy. After screening a panel of two drug …

    odu Repository record for Mechanisms of Cell Death Initiated in Herpes Simplex Virus Thymidine Kinase Expressing Colon Tumor Cells Treated with Ganciclovir and UCN-01 (opens in a new tab)

  3. An inducible, conditional and targeted B cell ablation mouse model for studying B cell functionality in the pathogenesis of human diseases

    … the herpes simplex virus 1 thymidine kinase (HSVtk) or diphtheria toxin (DT) receptor combined with their respective transgenic strategies. The ablation using HSVtk transgenic mice eliminates dividing cells, but does not affect non-dividing cells. In addition, because of its extremely high …

    temple Repository record for An inducible, conditional and targeted B cell ablation mouse model for studying B cell functionality in the pathogenesis of human diseases (opens in a new tab)

  4. Targeting The Blood-Brain Barrier With A Non-Canonical Iron-Mimicry Mechanism

    Treatment of central nervous system (CNS) diseases is limited by the blood-brain barrier (BBB), a selective vascular interface restricting passage of most molecules from blood into brain. Specific transport systems have evolved allowing circulating polar molecules to cross the BBB and gain access …

    uthsc Repository record for Targeting The Blood-Brain Barrier With A Non-Canonical Iron-Mimicry Mechanism (opens in a new tab)