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Showing 1 to 5 of 5 for “"Gads"”.

  1. Noncovalent Functionalization Of Graphene

    … a molecular footprint of 2.3 nm2 and a [DELTA]Gads = -38.8 ± 0.2 kJ mol-1. Its monolayers are stable in fresh electrolyte for more than 12 h and desorb from graphene 1000 times more slowly than model compounds bearing a single aromatic binding group. Tripods with naphthalene or phenanthrene …

    cornell Repository record for Noncovalent Functionalization Of Graphene (opens in a new tab)

  2. Comparison of CLEC-2 and GPVI signaling in platelets: the role of adaptor proteins

    … requirement for SLP-76 is not mediated by Gads. Both signalling pathways also show partial dependency for LAT. I also show that a novel protein, G6f, is not able to substitute for LAT in this signalling pathway and also exclude the LAT-family proteins PAG, LIME, LAX and NTAL as potential …

    birmingham Repository record for Comparison of CLEC-2 and GPVI signaling in platelets: the role of adaptor proteins (opens in a new tab)

  3. Interfacial electroactive assemblies: from molecular electronics to biological applications

    … isotherm, yielding a free energy of adsorption, △Gads, of -29 kJ.mol−1 for the Co2+ state and a limiting surface coverage 1.49 ± 0.25 x 10−11 mol.cm−2. The rate of electron transfer from the cobalt metal center to the electrode surface was found to be of the order of 1 x 105 s−1 by high speed …

    dcu Repository record for Interfacial electroactive assemblies: from molecular electronics to biological applications (opens in a new tab)

  4. Die Hämatopoetische Progenitor Kinase (HPK) 1 und NFAT-Transkriptionsfaktoren unterstützen die Apoptose von T-Lymphozyten

    … (LAT) und den Adaptorproteinen Nck, Crk, Gads, Grb2, Grap, CrkL sowie SLP-76 gezeigt. Für die Aktivierung der nach TZR-Stimulation in den Lipid-Rafts lokalisierten HPK1 sind sowohl Lck als auch ZAP-70 notwendig [Liou et al., 2000; Liu et al., 2000a; Ling et al., 2001]. Diese Daten legen …

    wurz-thes Repository record for Die Hämatopoetische Progenitor Kinase (HPK) 1 und NFAT-Transkriptionsfaktoren unterstützen die Apoptose von T-Lymphozyten (opens in a new tab)