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Showing 1 to 20 of 36 for “"Endocrinology and Diabetes"”.

  1. Cell coupling and exocytosis measured in intact mouse pancreatic islets Control of {delta}-cell secretion

    Patch-clamp and capacitance measurements were applied to α-, β- and δ-cells in intact mouse pancreatic islets. The maximum rate of β-cell exocytosis during a depolarization to 0 mV was 14 granules/s, <5% of that observed in isolated β-cells. β-cell exocytosis exhibited bell-shaped voltage …

    lund Repository record for Cell coupling and exocytosis measured in intact mouse pancreatic islets Control of {delta}-cell secretion (opens in a new tab)

  2. Exploring the Grey Zone between Type 1 and Type 2 Diabetes

    T1D is most common in children and young adults and is characterized by autoimmune destruction of insulin producing pancreatic beta cells, presence of certain risk genotypes such as HLA-DQB1, INS VNTR, PTPN22 and need of insulin for survival. In adults the same situation is often referred to as …

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  3. Genetics of Type 2 Diabetes and Metabolic Syndrome: From Genome Wide Linkage Scan and Candidate Genes to Genome Wide Association Studies

    Type 2 diabetes (T2D) and metabolic syndrome (MetS) are major health problems associated with cardiovascular disease. Both diseases are influenced by a combination of genetic and environmental factors. The aim of this thesis was to identify genetic risk factors for T2D, particularly T2D associated …

    lund Repository record for Genetics of Type 2 Diabetes and Metabolic Syndrome: From Genome Wide Linkage Scan and Candidate Genes to Genome Wide Association Studies (opens in a new tab)

  4. Regulation of Autoimmunity and Inflammation by microRNAs and Environmental Factors

    … is crucial for maintaining immunological balance and avoid autoimmune diseases like type 1 diabetes and the inflammatory bowel disease, ulcerative colitis (UC). In Study I, we have investigated the regulatory role of a class of noncoding RNAs, the miRNAs, during thymocyte development of the where …

    lund Repository record for Regulation of Autoimmunity and Inflammation by microRNAs and Environmental Factors (opens in a new tab)

  5. Outcome of Ischaemic Foot Ulcers in Patients with Diabetes, with or without Revascularization

    … in this thesis was to study patients with diabetes and severe peripheral arterial disease (PAD) in regard to outcomes of foot ulcers, factors related to outcomes, and occurrences of new ulcerations and amputations following healing of the initial ulcer. Patients with diabetes and a foot …

    lund Repository record for Outcome of Ischaemic Foot Ulcers in Patients with Diabetes, with or without Revascularization (opens in a new tab)

  6. Triggers of autoimmunity. Studies on gestational events.

    … factors is a risk factor for type 1 diabetes in the offspring. As children with type 1 diabetes have a higher risk of developing celiac disease (CD), an additional aim was to determine whether markers of possible infections during early pregnancy was associated with development of …

    lund Repository record for Triggers of autoimmunity. Studies on gestational events. (opens in a new tab)

  7. Hormone-Sensitive Lipase - New roles in adipose tissue biology

    … fat accumulation that may impair health, and increased knowledge about the enzymes controlling lipid metabolism is of great importance in order to combat this disease. Hormone-sensitive lipase (HSL) is a key enzyme in the mobilization of fatty acids from acylglycerols in adipocytes, but …

    lund Repository record for Hormone-Sensitive Lipase - New roles in adipose tissue biology (opens in a new tab)

  8. Inflammatory mediators in diabetic retinopathy

    … (DR) is the most feared complication of diabetes with an overall prevalence of 21.9-36.8% and may, if untreated, lead to severe visual disability or blindness. DR is histologically characterized by pericyte and endothelial cell loss, formation of acellular vessel strands, …

    lund Repository record for Inflammatory mediators in diabetic retinopathy (opens in a new tab)

  9. Mechanisms of defective insulin secretion in type 2 diabetes

    … B-cells is a central feature in type 2 diabetes (T2D). There is a strong hereditary component in type T2D, but the underlying pathophysiology remains largely unknown. This thesis uses a combination of gene network analysis and cell-physiological techniques to explore the genetic and

    lund Repository record for Mechanisms of defective insulin secretion in type 2 diabetes (opens in a new tab)

  10. Estrogen and Serotonin – old dogs, new tricks, Implications for pancreatic beta-cell function

    … regulated by metabolic status as well as local and circulating factors. These factors can activate different receptors on the pancreatic islet cells, for instance G-protein coupled receptors (GPCRs). When activated, these receptors are able to fine-tune islet hormone secretion and regulate …

    lund Repository record for Estrogen and Serotonin – old dogs, new tricks, Implications for pancreatic beta-cell function (opens in a new tab)

  11. Mechanisms underlying lipotoxicity and glucotoxicity in pancreatic islets

    … signal transduction pathways involved in FFA- and glucose-exerted effects on beta-cell function during both short-term as well as long-term exposure. The data presented here confirmed that the G-protein coupled receptor 40 (GPR40) is a functional FFA receptor expressed in pancreatic islets. We …

    lund Repository record for Mechanisms underlying lipotoxicity and glucotoxicity in pancreatic islets (opens in a new tab)

  12. β-cell dysfunction in Huntington's disease

    … is associated with an increased risk of diabetes mellitus. The reason for this is unknown, but considering the functional similarities of neurons and the insulin secreting pancreatic β-cell, pathological mechanisms may be shared by both cell types and account for neuronal as well as …

    lund Repository record for β-cell dysfunction in Huntington's disease (opens in a new tab)

  13. Studies in beta cells and adipocytes in the context of obesity and T2D - focusing on PDE3B, OPN and SCFAs

    Type 2 diabetes (T2D) is a heterogeneous disease characterized by altered lipid parameters and elevated glucose levels, as a direct consequence of impaired insulin signaling in target tissues and reduced insulin exocytosis from pancreatic β-cells. Obesity, which dramatically increases worldwide, is …

    lund Repository record for Studies in beta cells and adipocytes in the context of obesity and T2D - focusing on PDE3B, OPN and SCFAs (opens in a new tab)

  14. Genetic Loci Contributing to Spontaneous Autoimmune Diabetes

    Background and Aims: Spontaneous type 1 diabetes (T1D) in the BioBreeding (BB) rat mimics human T1D as the rats experience weight loss, polydipsia, polyuria, ketoacidosis, onset during puberty and insulin-dependency within a day after diagnosis. Because the DP rat develops T1D spontaneously, it is …

    lund Repository record for Genetic Loci Contributing to Spontaneous Autoimmune Diabetes (opens in a new tab)

  15. The role of genetic variation and DNA methylation in human glucose metabolism and type 2 diabetes

    The incidence of diabetes is increasing worldwide, with the most prevalent form being type 2 diabetes. Two fundamental processes contribute to the development of type 2 diabetes: insulin resistance in target organs and insufficient insulin secretion from the pancreatic beta-cells. The aim of this …

    lund Repository record for The role of genetic variation and DNA methylation in human glucose metabolism and type 2 diabetes (opens in a new tab)

  16. Genetic and epigenetic influence on oxidative phosphorylation, islet function and type 2 diabetes in humans

    The prevalence of type 2 diabetes (T2D) is increasing worldwide. T2D is a heterogeneous disease caused by a complex interplay between multiple genetic, epigenetic and non-genetic factors. The disease is characterised by impaired insulin secretion from pancreatic β-cells and insulin resistance in …

    lund Repository record for Genetic and epigenetic influence on oxidative phosphorylation, islet function and type 2 diabetes in humans (opens in a new tab)

  17. The Human Pancreatic Islet Methylome and Its Role in Type 2 Diabetes

    Islet dysfunction is central to the development and progression of type 2 diabetes (T2D). Epigenetic modifications are essential for establishing and maintaining cell identity and function in normal circumstances. Exposure to adverse environmental factors may alter the epigenome, and result in …

    lund Repository record for The Human Pancreatic Islet Methylome and Its Role in Type 2 Diabetes (opens in a new tab)

  18. Protein kinases in hormonal regulation of adipocyte metabolism.

    Abstract Along with liver and muscle tissue, adipose tissue helps maintain normal levels of glucose and lipids in the blood and has a very important role when it comes to storing lipids that can provide whole-body energy. After a meal is ingested, adipocytes take up glucose from the circulation and

    lund Repository record for Protein kinases in hormonal regulation of adipocyte metabolism. (opens in a new tab)

  19. The genetic background of gestational diabetes mellitus

    … determine the genetic background of gestational diabetes (GDM) and to examine how specific genes affect the development of diabetes post partum. In the DiPiS (Diabetes Prediction in Skåne) study we typed for HLA-DQB1 alleled, the transcription factor 7-like 2(TCF7L2) rs7903146, rs12255372 and

    lund Repository record for The genetic background of gestational diabetes mellitus (opens in a new tab)

  20. Candidate Genes for Late Diabetic Complications

    Diabetes mellitus is a group of metabolic diseases characterized by hyperglycemia resulting from defects in insulin secretion, insulin action, or both. The new WHO criteria for classification of diabetes takes into account also clinical stages dividing the diabetic patients into noninsulin …

    lund Repository record for Candidate Genes for Late Diabetic Complications (opens in a new tab)

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