Global ETD Search
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Showing 1 to 1 of 1 for “"Dioxin response element binding"”.
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Study of the aryl hydrocarbon receptor as a target for rational drug design
… D2 suppresses the 3-methylcholanthrene-induced, dioxin response element (DRE)-driven luciferase activity in Hep3B cells and exogenous Arnt reverses this D2 suppression. D2 suppresses the induction of CYP1A1 at both the message and protein levels in Hep3B cells; however, the CYP1B1 induction is …