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Showing 1 to 6 of 6 for “"DAPK"”.

  1. Control of DAPK-1 degradation

    Lin, YaoThe University of Edinburgh2009

    DAPK-1 is calcium-calmodulin regulated protein kinase involved in multiple cellular pathways including apoptosis, autophagy, cell survival and motility. The cytokine TNF-α has been reported to induce the degradation of DAPK-1. Here I identified the protease cathepsin B as a novel binding partner of …

    edinburgh Repository record

  2. Death- Associated Protein Kinase (DAPk); A Possible Modulator Of Neurodegeneration in A Caenorhabditis Elegans Model of Excittotoxicity

    Del Rosario, John Smith

    cuny Repository record

  3. Investigating the Roles of DAPK, p53/CEP-1, and Mitochondrial Damage in Necrotic Neurodegeneration in C. elegans

    Singh, Anil2024

    … such as the Death-Associated Protein Kinase (DAPK). However, the mechanism of DAPK’s action, and the possible involvement of downstream effectors like p53, mitochondria, and CypD, remain unclear. We use the geneticallyaccessible nematode C. elegans as a model organism to study excitotoxic …

    cuny Repository record

  4. The role of death-associated protein kinase in endothelial apoptosis under fluid shear stress

    Rennier, Keith R

    … and cancer. Death-associated protein kinase, DAPK is a pro-apoptotic regulator for various cell types that is localized in the cell cytoskeleton and regulates changes in cytoplasm associated with apoptosis. Yet its role in endothelial cells remains unclear. DAPK is a positive regulator in …

    purdue-thes Repository record

  5. DAPK3 Suppresses Mammary Acini Morphogenesis and is Required for Mouse Development

    Kocher, Brandon2016

    … several lines of research have indicated that DAPK3, a member of the serine/threonine death associated protein kinase (DAPK) family, plays an important role in various signaling pathways important to tissue homeostasis and mammalian biology. Considered to be a putative tumor suppressor, the …

    wustl Repository record

  6. Analysis of prognostic and drug resistance markers in lung cancer

    Davidson, Scott MitchellUniversity of Glasgow2007

    … lung tissue. The markers studied were MLH1, p16, DAPK, TIMP 3, HIC 1, MINT 25, MINT 31 and RASSF1A. In this study 30 (60%) of samples exhibited methylation of at least one promoter site with 19 (38%) at 1 site, 5 (10%) at 2 sites, 2 (4%) at 3 sites and 4 (8%) at 4 sites. Twenty (40%) of the tumour …

    glasgow Repository record