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Showing 1 to 16 of 16 for “"Cerebral edema"”.

  1. The effect of calcium channel blocking agents on cold induced cerebral edema in mice

    This document only includes an excerpt of the corresponding thesis or dissertation. To request a digital scan of the full text, please contact the Ruth Lilly Medical Library's Interlibrary Loan Department (rlmlill@iu.edu).

    iupui Repository record for The effect of calcium channel blocking agents on cold induced cerebral edema in mice (opens in a new tab)

  2. Evaluation & design of a novel drug delivery device for treating tumor-related cerebral edema

    Tumor-related cerebral edema is a debilitating medical condition that afflicts tens of thousands of newly diagnosed brain cancer patients in the U.S. each year, where the standard care of treatment indicates the systemic administration of dexamethasone sodium phosphate (DXM) and surgery. The former …

    mit Repository record for Evaluation & design of a novel drug delivery device for treating tumor-related cerebral edema (opens in a new tab)

  3. The role of substance P in cerebral edema associated with rat and human infarction and contusion

    … both traumatic brain injury (TBI) and stroke is cerebral edema, but its mechanism is poorly understood. Recent evidence suggests that substance P (SP) plays a role in this edema and that injured SP-containing perivascular nerve fibres are involved. A study which examined tissue obtained from rat …

    adelaide Repository record for The role of substance P in cerebral edema associated with rat and human infarction and contusion (opens in a new tab)

  4. A Study of Measuring Intracranial Pressure Using a Non-Invasive Piezoelectric Sensor

    … body, can experience swelling, also known as cerebral edema. Cerebral edema may occur because of an injury, health related issues, tumors, or even high altitudes[1]. When cerebral edema occurs, a rise in intracranial pressure (ICP) becomes prevalent and may cause a serious threat. Without …

    vt Repository record for A Study of Measuring Intracranial Pressure Using a Non-Invasive Piezoelectric Sensor (opens in a new tab)

  5. Oxygen resuscitation does not ameliorate neonatal hypoxia/ischemia-induced edema

    … and behavioral deficits in children. Although cerebral edema is a common outcome after HI, the mechanisms leading to the excessive fluid accumulation are poorly understood. Hyperoxia treatment after HI (HHI) is the recommended clinical therapy for newborn resuscitation. Our objective was to …

    utmb Repository record for Oxygen resuscitation does not ameliorate neonatal hypoxia/ischemia-induced edema (opens in a new tab)

  6. Peripheral Leukocytes and Intracerebral Hemorrhage

    … significant disability from spontaneous intracerebral hemorrhage (ICH) every year. No proven treatment is available that improves ICH outcomes. Since hematoma volume is the strongest predictor of ICH outcome, limiting hematoma expansion has been the primary goal of recent ICH clinical trials. …

    ohiolink Repository record for Peripheral Leukocytes and Intracerebral Hemorrhage (opens in a new tab)

  7. Bromethalin-based rodenticides: Mode of action, toxicity, clinical effects, and treatment efficacy in rats, dogs, and cats

    … with increased cerebrospinal fluid pressure and cerebral edema. Bromethalin toxicosis produces acute and chronic EEG changes. Predominant abnormal EEG changes included: spike and spike-and-wave EEG patterns; high voltage slow wave activity; photoconvulsive or photoparoxysmal irritative responses, …

    uiuc Repository record for Bromethalin-based rodenticides: Mode of action, toxicity, clinical effects, and treatment efficacy in rats, dogs, and cats (opens in a new tab)

  8. Η συμβολή της ηλεκτρονικής τομογραφίας στη διαφοροδιάγνωση των βλαβών του ενδοκρανίου στην οξεία φάση των κρανιοεγκεφαλικών κακώσεων

    … in ideal method in estimating the concomitant cerebral edema and the shift of midline structures, which are crucial as far as the severity of the injury and the clinical progress of the patient, are concerned. It is also a valuable follow-up method after surgical intervention or conservative …

    greece Repository record for Η συμβολή της ηλεκτρονικής τομογραφίας στη διαφοροδιάγνωση των βλαβών του ενδοκρανίου στην οξεία φάση των κρανιοεγκεφαλικών κακώσεων (opens in a new tab)

  9. First-In-DOg HISTotripsy for Intracranial Tumors Trial: The FIDOHIST Study

    … an adverse event consisting of transient cerebral edema that was possibly attributable to histotripsy. Histotripsy ablations could be grossly visualized and identified on MRI, with features consistent with hemorrhage and necrosis. Significant expression or upregulation of the damage …

    vt Repository record for First-In-DOg HISTotripsy for Intracranial Tumors Trial: The FIDOHIST Study (opens in a new tab)

  10. The Expression and Cellular Localization of Cc-Chemokine Receptor 5 (Ccr5) After Traumatic Brain Injury

    … can include scalp laceration, skull fraction, cerebral contusions and lacerations as well as intracranial hemorrhage. Following the initial insult, a delayed response occurs and is characterized by hypoxia, ischemia, cerebral edema, and infection. During secondary brain injury, a series of …

    uthsc Repository record for The Expression and Cellular Localization of Cc-Chemokine Receptor 5 (Ccr5) After Traumatic Brain Injury (opens in a new tab)

  11. The Expression and Cellular Localization of Cc-Chemokine Receptor 5 (Ccr5) After Traumatic Brain Injury

    … can include scalp laceration, skull fraction, cerebral contusions and lacerations as well as intracranial hemorrhage. Following the initial insult, a delayed response occurs and is characterized by hypoxia, ischemia, cerebral edema, and infection. During secondary brain injury, a series of …

    uthsc Repository record for The Expression and Cellular Localization of Cc-Chemokine Receptor 5 (Ccr5) After Traumatic Brain Injury (opens in a new tab)

  12. Caveolins and NJKs Influence Brain Endothelial Permeability after Juvenile TBI

    … resulting in increased brain swelling, disrupted cerebral blood flow (CBF) autoregulation, long-term disabilities and death following TBI in young demographic. Unsurprisingly, BBB disruption and the resultant cerebral edema have emerged as therapeutic targets in juvenile TBI. It is therefore …

    loma-linda Repository record for Caveolins and NJKs Influence Brain Endothelial Permeability after Juvenile TBI (opens in a new tab)

  13. Point-of-care ultrasound abnormalities in late onset severe preeclampsia: prevalence and association with serum albumin and brain natriuretic peptide

    … indicate the presence of pulmonary interstitial edema, cerebral edema, and cardiac dysfunction. Laboratory markers of oncotic pressure (albumin) and cardiac dysfunction (brain natriuretic peptide [BNP]) may be abnormal, but the clinical application remains unclear. We investigated the prevalence …

    cape-town Repository record for Point-of-care ultrasound abnormalities in late onset severe preeclampsia: prevalence and association with serum albumin and brain natriuretic peptide (opens in a new tab)

  14. The Role of Adenosine Signaling In Mature Erythrocytes and Erythroid Progenitors

    … intervention, it can progress to pulmonary and cerebral edema, stroke, cardiovascular dysfunction and death. Although hypoxia-induced tissue damage is a serious condition with high morbidity and mortality, safe and effective mechanism-based therapies are limited.</p> <p>The erythrocyte is the …

    uthsc Repository record for The Role of Adenosine Signaling In Mature Erythrocytes and Erythroid Progenitors (opens in a new tab)

  15. Hyperinflammatory Experimental Cerebral Malaria: Coagulation and Glial Cell Activation in Plasmodium chabaudi Infection of IL-10-deficient Mice

    Cerebral malaria is one of the most severe complications of Plasmodium falciparum infection and occurs mostly in young African children. This syndrome results from a combination of high levels of parasitemia and inflammation. While mutations in inflammatory cytokine genes and the levels of …

    utmb Repository record for Hyperinflammatory Experimental Cerebral Malaria: Coagulation and Glial Cell Activation in Plasmodium chabaudi Infection of IL-10-deficient Mice (opens in a new tab)

  16. MAPK and pro-inflammatory mediators in the walls of brain blood vessels following cerebral ischemia

    … an important role in the development of the cerebral lesions [6]. This inflammatory reaction involves activation of resident cells (mainly microglia), infiltration and accumulation of various inflammatory cells (including neutrophils, leukocytes, monocytes, macrophages), and production of …

    lund Repository record for MAPK and pro-inflammatory mediators in the walls of brain blood vessels following cerebral ischemia (opens in a new tab)