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Showing 1 to 7 of 7 for “"CIMP"”.

  1. Impact of the gut microbiota on DNA methylation in colorectal cancer

    The CpG Island Methylator Phenotype (CIMP) is a distinct form of aberrant DNA methylation in cancer, and it is seen in 20-40% of colorectal cancers (CRC) where its causes remain elusive. Intestinal microbiota represents an important environmental component implicated in CRC development. …

    temple Repository record for Impact of the gut microbiota on DNA methylation in colorectal cancer (opens in a new tab)

  2. Clinical importance of microsatellite instability and promoter methylation of DNA in colorectal cancer // Клинично значение на микросателитната нестабилност и промоторното метилиране на ДНК при колоректален карцином

    … продукция на муцин и лимфоцитна реакция. CIMP-фенотипът се асоциира с напреднала възраст, проксимална локализация, ниска степен на диференциация и инфилтрация в чревната стена. По-ниска преживяемост е налице при туморите с метилиран hMLH, CIMP-фенотип и секреция на муцин. Адювантната …

    varna Repository record for Clinical importance of microsatellite instability and promoter methylation of DNA in colorectal cancer // Клинично значение на микросателитната нестабилност и промоторното метилиране на ДНК при колоректален карцином (opens in a new tab)

  3. Rna-Sequencing Applications: Gene Expression Quantification and Methylator Phenotype Identification

    … to identify the CpG island methylator phenotype (CIMP) by integrating datasets of mRNA expression level and DNA methylation status.</p> <p>Section 1: The cost of DNA sequencing has reduced dramatically in the past decade. Consequently, genomic research increasingly depends on sequencing …

    uthsc Repository record for Rna-Sequencing Applications: Gene Expression Quantification and Methylator Phenotype Identification (opens in a new tab)

  4. Το μικροβίωμα του παχέος εντέρου στην καρκινογέννεση. Κλινική αξία στην πρόγνωση και θεραπεία της νόσου

    … (MSI) και του φαινοτύπου μεθυλίωσης CpG νησίδων (CIMP). Αυτές οι οδοί οδηγούν σε συσσώρευση γενετικών και επιγενετικών αλλοιώσεων που προάγουν τη μετατροπή φυσιολογικού επιθηλίου σε καρκινικό. Το εντερικό μικροβίωμα διαδραματίζει καθοριστικό ρόλο τόσο στη φυσιολογία όσο και στην παθογένεση του …

    athens Repository record for Το μικροβίωμα του παχέος εντέρου στην καρκινογέννεση. Κλινική αξία στην πρόγνωση και θεραπεία της νόσου (opens in a new tab)

  5. EBV-Associated Gastric Cancer: From Initial Infection to Unique Therapeutic Approaches

    … known as a CpG island hypermethylator phenotype (CIMP). EBV infection of B cells can be easily modeled in vitro using primary B cells and lymphoblastoid cell lines (LCLs). However, epithelial cell infection models have proven much more difficult to develop. Given these difficulties, EBV infection …

    duke Repository record for EBV-Associated Gastric Cancer: From Initial Infection to Unique Therapeutic Approaches (opens in a new tab)

  6. The Role of Epigenetics in Hereditary Colorectal Cancer Tumorigenesis: Validation of Genome-Wide Methylation Array Results through Bisulfite Sequencing

    Colorectal cancer (CRC) poses a considerable global health challenge, with high mortality rates despite advancements in cancer research. Approximately one in ten CRC cases have a hereditary basis, with Lynch syndrome and Familial adenomatous polyposis standing as the two predominant …

    helsinki Repository record for The Role of Epigenetics in Hereditary Colorectal Cancer Tumorigenesis: Validation of Genome-Wide Methylation Array Results through Bisulfite Sequencing (opens in a new tab)

  7. Epigenetic Modification as a Therapeutic Target in BRAFV600E-mutated Metastatic Colorectal Cancer

    <p>Patients with BRAF<sup>V600E</sup>-mutated metastatic colorectal cancer (mCRC) experience a worse prognosis and demonstrate only a 5% response rate to BRAF inhibitor treatment. In this study, adaptive resistance, and a potential combination of standard therapies in BRAF<sup>V600E</sup> CRC were …

    uthsc Repository record for Epigenetic Modification as a Therapeutic Target in BRAFV600E-mutated Metastatic Colorectal Cancer (opens in a new tab)