Global ETD Search

Search theses and dissertations gathered from participating repositories worldwide. Every result links back to the library that holds it. No account is needed.

Results

Showing 1 to 3 of 3 for “"CD155"”.

  1. Exploring Veto Activity: DNAM-1-CD155 Axis in Overcoming NK Cell-Mediated Allo-Rejection and Applications for CAR-T Cell Therapy

    … also discovered that veto Tcm cells upregulate CD155, a primary ligand for the activating receptor DNAM-1. Later in vitro studies further indicated that conjugation between veto Tcm cells and alloreactive NK cells induces NK cell anergy by enhancing the internalization and degradation of DNAM-1, …

    uthsc Repository record for Exploring Veto Activity: DNAM-1-CD155 Axis in Overcoming NK Cell-Mediated Allo-Rejection and Applications for CAR-T Cell Therapy (opens in a new tab)

  2. Delivery of sting agonist using lipid nanoparticles and discovery of anti-TIGIT peptides for cancer therapy

    … the receptor TIGIT and its high-affinity ligand, CD155. In clinical studies, inhibiting this pathway within the adaptive immune system has demonstrated the ability to reverse the exhaustion of T- and NK cells, thereby restoring their functional ability to elicit cytotoxic activity against tumor …

    umkc Repository record for Delivery of sting agonist using lipid nanoparticles and discovery of anti-TIGIT peptides for cancer therapy (opens in a new tab)

  3. Effect of Entinostat On Nk Cell-Mediated Cytotoxicity Against Os Cells and Os Lung Metastsis

    … on OS cells (MIC A/B, ULBP1, ULBP2/5/6, and CD155) and enhanced the NK cell-mediated cytotoxicity. Entinostat oral administration also increased MICA/B expression on lung tumors. Entinostat (≤ 2 μM) did not have any adverse effect on NK cell viability, receptor expression, or function within …

    uthsc Repository record for Effect of Entinostat On Nk Cell-Mediated Cytotoxicity Against Os Cells and Os Lung Metastsis (opens in a new tab)