Global ETD Search

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Showing 1 to 4 of 4 for “"CASP4"”.

  1. Inflammasome upregulation and activation in photoreceptors of type 2 diabetes and N-methyl-N-nitrosourea models is attenuated by Wedelolactone

    … canonical (CASP1) and non-canonical pathways (CASP4/11). Oxidative stress plays an important role in vascular and neurodegenerative changes in DR and has been shown to induce mitochondrial dysfunction in various retinal cells where it can manifest as loss of organelle membrane integrity and …

    qu-belfast Repository record for Inflammasome upregulation and activation in photoreceptors of type 2 diabetes and N-methyl-N-nitrosourea models is attenuated by Wedelolactone (opens in a new tab)

  2. Entamoeba histolytica-induced Activation of Caspase-4 Regulates Gasdermin D Cleavage to Mediate IL-1β Secretion in Macrophages

    … cytokine release, CRISPR-Cas9 gene editing of CASP4/1, ASC, NLRP3, GSDMD THP-1 cells were utilized. In the absence of caspase-1, caspase-4 activation was significantly upregulated and enhanced GSDMD pore formation to trigger robust IL-1β secretion as quantified by HEK-BlueTM reporter cell. The …

    calgary Repository record for Entamoeba histolytica-induced Activation of Caspase-4 Regulates Gasdermin D Cleavage to Mediate IL-1β Secretion in Macrophages (opens in a new tab)

  3. The role of IFN-γ in cell-autonomous immune responses against Salmonella Typhimurium and Toxoplasma gondii

    The research presented in this thesis investigates the function of interferon gamma in vacuoles, namely Salmonella enterica serovar Typhimurium and Toxoplasma gondii. IFN-γ is a cytokine that functions in both the innate and the adaptive immune responses. IFN-γ is mainly secreted by T cells and …

    cambridge Repository record for The role of IFN-γ in cell-autonomous immune responses against Salmonella Typhimurium and Toxoplasma gondii (opens in a new tab)

  4. Differential proteomic profiling towards elucidation of TB-IRIS pathogenesis

    … stress and overactivation (including via NLRC4, CASP4/5, TLR/IL-1R-MyD88-NFκB, and MAPK-AP1 signalling), producing - instead of Mtb clearance - inflammatory cell death with release of immune-activating and tissue-damaging host- and Mtb-derived molecules. Reactivation of Mtb lymphocyte memory …

    cape-town Repository record for Differential proteomic profiling towards elucidation of TB-IRIS pathogenesis (opens in a new tab)