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Showing 1 to 20 of 22 for “"Amyloid fibril"”.

  1. Amyloid Fibril Nucleation In Reverse Micelles

    The 40-residue amyloid beta protein (Abeta) is the unstructured cleavage product of a common membrane protein that is produced in large quantities, but normally cleared from the brain before it exerts any apparent toxicity. Under some conditions, however, it undergoes a conformational change and …

    penn Repository record for Amyloid Fibril Nucleation In Reverse Micelles (opens in a new tab)

  2. Amyloid fibril formation in Alzheimer's disease

    Thesis (Ph. D.)--Massachusetts Institute of Technology, Dept. of Chemistry, 1993.

    mit Repository record for Amyloid fibril formation in Alzheimer's disease (opens in a new tab)

  3. Amyloid fibril structure of peptides and proteins by magic angle spinning NMR spectroscopy and dynamic nuclear polarization

    Amyloid fibrils are insoluble, non-crystalline protein filaments associated with a number of diseases such as Alzheimer's and Type Il diabetes. They can have a functional role in different organisms and many proteins and peptides have been found to form amyloid fibrils in vitro. We have used magic …

    mit Repository record for Amyloid fibril structure of peptides and proteins by magic angle spinning NMR spectroscopy and dynamic nuclear polarization (opens in a new tab)

  4. Thermodynamic Characterisation of Amyloid Fibrils

    Amyloid fibril related diseases include dementia, Alzheimer’s disease and Parkinsons disease and pose an increasingly large burden to global healthcare, due to the presence of an ageing population. Despite many healthcare advances, amyloid fibril diseases remain largely untreatable, with only …

    cambridge Repository record for Thermodynamic Characterisation of Amyloid Fibrils (opens in a new tab)

  5. THE ROLE OF BACTERIAL AMYLOID FIBRILS IN ESCHERICHIA COLI COMPLEMENT RESISTANCE

    … to the complement system. The bacterial amyloid fibril, curli, functions in bacterial adherence and the formation of biofilm. Curli-producing parental and curli-deficient mutant E. coli was compared in its survival to human complement, using in vitro serum sensitivity assays. Results …

    temple Repository record for THE ROLE OF BACTERIAL AMYLOID FIBRILS IN ESCHERICHIA COLI COMPLEMENT RESISTANCE (opens in a new tab)

  6. Solid-state NMR studies of α-synuclein fibril structure

    … component, α-synuclein (α- syn), adopts an amyloidogenic fibrillar form in Lewy bodies. In recent years research has slowly started to unravel clear pathological links between this fibrillar protein and PD disease progression. While great progress is being made, it has become evident that …

    uiuc Repository record for Solid-state NMR studies of α-synuclein fibril structure (opens in a new tab)

  7. The Role of Force-Sensitive Interactions in Amyloid Assembly by Fungal Adhesins

    … segment of their sequence, termed the amyloid-forming region (AFR). At the core of these adhesin aggregates are parallel or anti-parallel β-sheet arrays, which can layer onto other adhesin β-sheets by sidechain–sidechain interactions to form highly stable amyloid fibrils. The Als …

    queens Repository record for The Role of Force-Sensitive Interactions in Amyloid Assembly by Fungal Adhesins (opens in a new tab)

  8. Intracellular peptide library screening to derive inhibitors of Parkinson's disease associated α-synuclein aggregation

    Aggregation of α-synuclein (α-syn) into toxic fibrils is a pathogenic hallmark of Parkinson’s disease (PD). This research aimed to develop peptides capable of inhibiting α-syn aggregation using a semi-rational design combined with a multiplexed intracellular Protein-fragment Complementation Assay …

    essex Repository record for Intracellular peptide library screening to derive inhibitors of Parkinson's disease associated α-synuclein aggregation (opens in a new tab)

  9. On the Kinetics of Protein Misfolding and Aggregation

    Protein (mis)folding into highly ordered, fibrillar structures, amyloid fibrils, is a hallmark of several, mainly neurodegenerative, disorders. The mechanism of this supra-molecular self-assembly reaction, as well as its relationship to protein folding are not well understood. In particular, the …

    cambridge Repository record for On the Kinetics of Protein Misfolding and Aggregation (opens in a new tab)

  10. Structure and dynamics of membrane proteins from solid-state NMR

    … coordination geometry of a de novo designed amyloid fibril that catalyzes ester hydrolysis. By measuring the intermolecular contacts, we determined that peptides form parallel-in- register P-sheets and further assemble into stacked bilayers in an antiparallel orientation. The zinc binding …

    mit Repository record for Structure and dynamics of membrane proteins from solid-state NMR (opens in a new tab)

  11. Order, disorder, and protein aggregation

    … process, from early nucleation events to fibril elongation. In the first study, I present a conformational ensemble of a-synuclein, the culprit protein of Parkinson's disease, constructed using a Variational Bayesian Weighting algorithm in combination with NMR data collected by our …

    mit Repository record for Order, disorder, and protein aggregation (opens in a new tab)

  12. Unzipping Amyloid Fibrils: How a Novel Calcium-Binding Protein, NUCB1, Prevents the Formation of Amyloid Fibrils

    … we also established novel and unique anti-amyloidogenic functional ability of sNUCB1. We show that Ca<sup>2+</sup>-free sNUCB1 can inhibit fibril formation by highly amyloidogenic human Islet Amyloid PolyPeptide (hIAPP) and Amyloid-β 42 (Aβ42) peptides, as relevant to Type-2 Diabetes and …

    rockefeller Repository record for Unzipping Amyloid Fibrils: How a Novel Calcium-Binding Protein, NUCB1, Prevents the Formation of Amyloid Fibrils (opens in a new tab)

  13. Investigation Into Protein Folding and Misfolding

    … structure and the second is misfolding into an amyloid fibril structure. In the first-half of this work, we investigated the folding of the BI domain of the <em>Streptococcal</em> immunoglobulin-binding domain of protein G (GB1) as our model system. Using bioinformatics approaches we …

    odu Repository record for Investigation Into Protein Folding and Misfolding (opens in a new tab)

  14. Studies on the physical stability of a C-terminally amidated variant of GLP-1

    … species which then further elongate to form amyloid fibrils. When a peptide aggregates, it leads not only to the loss of its biological activity but it can also give rise to other critical problems, e.g. toxicity and immunogenicity, associated with the formation of intermediate oligomeric …

    cambridge Repository record for Studies on the physical stability of a C-terminally amidated variant of GLP-1 (opens in a new tab)

  15. An interdisciplinary approach to studying mechanistic, structural and toxic features of protein aggregates associated with neurodegenerative disorders.

    … with protein aggregation have been attributed to amyloidogenic species that are present during the misfolding process. In particular, oligomeric species are, however, intrinsically difficult to study as a consequence of their low abundance and highly heterogeneous nature. The first chapter of my …

    cambridge Repository record for An interdisciplinary approach to studying mechanistic, structural and toxic features of protein aggregates associated with neurodegenerative disorders. (opens in a new tab)

  16. Smart Nanomaterials from Repeat Proteins and Amyloid Fibrils

    … components for protein-based materials are amyloid fibrils and tandem repeat proteins. Amyloid fibrils are exceptionally strong, tough, highly-ordered structures that self-assemble from a wide range of simple building blocks. Meanwhile, tandem repeat proteins are a class of proteins that act …

    cambridge Repository record for Smart Nanomaterials from Repeat Proteins and Amyloid Fibrils (opens in a new tab)

  17. Structural Polymorphism of Alpha-Synuclein in Conditions Resembling the Cellular Environment

    … (aSyn), from its functional disordered form into amyloid fibrils with characteristic β-sheet structure, as being at the centre of PD. However, many unanswered questions remain with regards to the aSyn aggregation mechanism in neurons and, in particular, the earliest steps of this process, when the …

    cambridge Repository record for Structural Polymorphism of Alpha-Synuclein in Conditions Resembling the Cellular Environment (opens in a new tab)

  18. COMPUTATIONAL MODELLING OF PROTEIN FIBRILLATION WITH APPLICATION TO GLUCAGON

    … method to model the steric zipper of amyloid fibrils (FibPreditor) is developed. The method generates an ensemble of structures for the steric zipper by a number of geometric operations and presents the most energetically favorable candidates as models of steric zipper. The method is …

    purdue-thes Repository record for COMPUTATIONAL MODELLING OF PROTEIN FIBRILLATION WITH APPLICATION TO GLUCAGON (opens in a new tab)

  19. Self-assembly of a gonadotropin-releasing hormone antagonist-Teverelix

    … under other conditions it was known to form fibrillar structures. The mechanism of formation of either state, and the factors affecting the stability and rate of formation of these states was largely unknown. Since the behaviour of Tv at low and high concentrations is significantly different, …

    cambridge Repository record for Self-assembly of a gonadotropin-releasing hormone antagonist-Teverelix (opens in a new tab)

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