Global ETD Search
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Showing 1 to 4 of 4 for “"AGE-RAGE"”.
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A ROLE FOR ANGIOTENSIN-(1-7) TO ATTENUATE ADVANCED GLYCATION END PRODUCT -INDUCED MYOFIBROBLAST TRANSITION IN THE NRK-52E RENAL PROXIMAL TUBULE CELL LINE
… tubules cells. Advanced glycated end product (AGEs) including methylglyoxal-modified albumin (MGA) may contribute to the development and progression of diabetic nephropathy via activation of the receptors for AGE (RAGE) and the RAS. The RAS is functionally partitioned into the angiotensin II …
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Endocrine, cellular and molecular effects of diabetes mellitus on male fertility
… similar, sperm nuclear and mitochondrial DNA damage was significantly higher in diabetic men. This suggests that exposure to the diabetic environment is associated with damage to sperm at a molecular but not a cellular level.<br/><br/>Subsequently, oxidative DNA damage was assessed using High …
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Protective effects of regular exercise and Korea red ginseng against high-fat diet-increased renal injury in ᴅ-galactose-induced aging rats
… Aging is progressive accumulation of oxidative agents. Advanced glycation end products (AGEs) and advanced lipoxidation end products (ALEs) formation has been implicated in the aging process. Obesity induced by a high-fat diet (HFD) may reduce renal function. However, the impact of obese on the …